BCL-2 and BOK regulate apoptosis by interaction of their C-terminal transmembrane domains.

Autor: Beigl TB; Robert Bosch Center for Tumor Diseases, Stuttgart, Germany., Paul A; Robert Bosch Center for Tumor Diseases, Stuttgart, Germany., Fellmeth TP; Cluster of Excellence SimTech, University of Stuttgart, Stuttgart, Germany., Nguyen D; Department of Medical Biophysics, Faculty of Medicine, University of Toronto, Toronto, Canada.; Biological Sciences Platform, Sunnybrook Research Institute, Toronto, Canada., Barber L; Robert Bosch Center for Tumor Diseases, Stuttgart, Germany., Weller S; Robert Bosch Center for Tumor Diseases, Stuttgart, Germany., Schäfer B; Robert Bosch Center for Tumor Diseases, Stuttgart, Germany., Gillissen BF; Department of Hematology, Oncology, and Tumorimmunology, Charité University Medicine, Berlin, Germany., Aulitzky WE; Robert-Bosch-Hospital, Stuttgart, Germany., Kopp HG; Robert Bosch Center for Tumor Diseases, Stuttgart, Germany.; Robert-Bosch-Hospital, Stuttgart, Germany., Rehm M; Institute of Cell Biology and Immunology, University of Stuttgart, Stuttgart, Germany.; Stuttgart Research Center Systems Biology, University of Stuttgart, Stuttgart, Germany., Andrews DW; Department of Medical Biophysics, Faculty of Medicine, University of Toronto, Toronto, Canada.; Biological Sciences Platform, Sunnybrook Research Institute, Toronto, Canada.; Department of Biochemistry, Faculty of Medicine, University of Toronto, Toronto, Canada., Pluhackova K; Cluster of Excellence SimTech, University of Stuttgart, Stuttgart, Germany., Essmann F; Robert Bosch Center for Tumor Diseases, Stuttgart, Germany. frank.essmann@bosch-health-campus.com.; Department of Molecular Medicine, Interfaculty Institute for Biochemistry, Eberhard Karls University Tübingen, Tübingen, Germany. frank.essmann@bosch-health-campus.com.

Publikováno v: EMBO reports [EMBO Rep] 2024 Sep; Vol. 25 (9), pp. 3896-3924. Date of Electronic Publication: 2024 Jul 24.