Výsledky vyhledávání - "Felicia A, Hunter"

Autor: Binu, Tharakan, Felicia A, Hunter, Saravanakumar, Muthusamy, Sonya, Randolph, Crystal, Byrd, Veena N, Rao, E Shyam P, Reddy, Ed W, Childs

Publikováno v: Shock. 57:309-315

ERG (ETS-related gene) is a member of the ETS (Erythroblast-transformation specific) family of transcription factors abundantly present in vascular endothelial cells. Recent studies demonstrate that ERG has important roles in blood vessel stability a

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::59a609b0207c96c8c6e93e086dbb764e
https://doi.org/10.1097/shk.0000000000001899

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Protective effects of FK 506 against haemorrhagic shock-induced microvascular hyperpermeability

Autor: Binu Tharakan, Ed W. Childs, Felicia A. Hunter

Publikováno v: Clinical and experimental pharmacologyphysiologyREFERENCES. 48(12)

Microvascular hyperpermeability, the excessive leakage of fluid and proteins from the intravascular space to the interstitium, is a devastating clinical concern in haemorrhagic shock (HS), sepsis, burn and so forth. Previous studies have shown that H

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2b6520f3a5612b0e20ac0af587331016
https://pubmed.ncbi.nlm.nih.gov/34432902

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Burn Injury-Associated MHCII+ Immune Cell Accumulation Around Lymphatic Vessels of the Mesentery and Increased Lymphatic Endothelial Permeability Are Blocked by Doxycycline Treatment

Autor: Felicia A. Hunter, Ed W. Childs, Karen M. Doersch, Walter E. Cromer, Scott D. Zawieja, Hayden W. Stagg, David C. Zawieja, Binu Tharakan

It is theorized that toxic agents are transported from the hyperpermeable gut of burn victims through the lymph, to the systemic circulation, causing global injury. We believe that immune cells respond to leakage of "toxic lymph" following trauma cau

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6408edf1758bd32bb2703c984bcf541a
https://europepmc.org/articles/PMC5810432/

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Glycogen synthase kinase 3 inhibitor protects against microvascular hyperpermeability following hemorrhagic shock

Autor: Felicia A. Hunter, Ed W. Childs, Devendra A. Sawant, Binu Tharakan

Publikováno v: Journal of Trauma and Acute Care Surgery. 79:609-616

BACKGROUND Hemorrhagic shock (HS)-induced microvascular hyperpermeability involves disruption of endothelial cell adherens junctions leading to increase in paracellular permeability. β-Catenin, an integral component of the adherens junctional comple

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::987f3d203dc13aa1fb7715c781fc6aac
https://doi.org/10.1097/ta.0000000000000807

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Regulation of tumor necrosis factor-α–induced microvascular endothelial cell hyperpermeability by recombinant B-cell lymphoma-extra large

Autor: Rickesha L. Wilson, Ed W. Childs, Felicia A. Hunter, Binu Tharakan, Devendra A. Sawant, Hayden W. Stagg

Publikováno v: Journal of Surgical Research. 184:628-637

Background Tumor necrosis factor-α (TNF-α), a cytotoxic cytokine, induces endothelial cell barrier dysfunction and microvascular hyperpermeability, leading to tissue edema, a hallmark of traumatic injuries. The objective of the present study was to

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3319ec6d21a45b7c2362710b9311864b
https://doi.org/10.1016/j.jss.2013.04.079

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Inhibition Of Fas–fas Ligand Interaction Attenuates Microvascular Hyperpermeability Following Hemorrhagic Shock

Autor: Felicia A. Hunter, W. Roy Smythe, Hayden W. Stagg, Ed W. Childs, Devendra A. Sawant, Binu Tharakan, Richard P Tobin, M. Karen Newell

Publikováno v: Shock. 39:161-167

Hemorrhagic shock (HS)-induced microvascular hyperpermeability poses a serious challenge in the management of trauma patients. Microvascular hyperpermeability occurs mainly because of the disruption of endothelial cell adherens junctions, where the "

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::126494e87b9a6d1bb0aaab0ca51f88a5
https://doi.org/10.1097/shk.0b013e31827bba73

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