Zobrazeno 1 - 10
of 16
pro vyhledávání: '"Fedor Nikulenkov"'
RAD51 separation of function mutation disables replication fork maintenance but preserves DSB repair
Autor:
Mi Young Son, Ondrej Belan, Mario Spirek, Jakub Cibulka, Fedor Nikulenkov, You Young Kim, Sunyoung Hwang, Kyungjae Myung, Cristina Montagna, Tae Moon Kim, Lumir Krejci, Paul Hasty
Publikováno v:
iScience, Vol 27, Iss 4, Pp 109524- (2024)
Summary: Homologous recombination (HR) protects replication forks (RFs) and repairs DNA double-strand breaks (DSBs). Within HR, BRCA2 regulates RAD51 via two interaction regions: the BRC repeats to form filaments on single-stranded DNA and exon 27 (E
Externí odkaz:
https://doaj.org/article/9292610b2dda480b8b8912a6757729c2
Autor:
Victoria Marini, Fedor Nikulenkov, Pounami Samadder, Sissel Juul, Birgitta R. Knudsen, Lumir Krejci
Publikováno v:
BMC Biology, Vol 21, Iss 1, Pp 1-16 (2023)
Abstract Background DNA-protein cross-links (DPCs) are one of the most deleterious DNA lesions, originating from various sources, including enzymatic activity. For instance, topoisomerases, which play a fundamental role in DNA metabolic processes suc
Externí odkaz:
https://doaj.org/article/f27960dd49134ebbb90288484d64163e
Autor:
Karel Souček, Kamil Paruch, Lumír Krejčí, Aleš Hampl, Josef Jaroš, Milan Ešner, Radek Fedr, Peter Kollár, Petra Ovesná, Miroslava Bittová, Lucia Binó, Hana Kolářová, Kateřina Vašíčková, Petr Vaňhara, Nicol Straková, Stanislav Drápela, Fedor Nikulenkov, Prashant Khirsariya, Ondřej Hylse, Tereza Suchánková, Pounami Samadder
Detail description of synthesis and supporting data
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::621a022e094e2d4ba0700755c1671825
https://doi.org/10.1158/1535-7163.22508934
https://doi.org/10.1158/1535-7163.22508934
Autor:
Karel Souček, Kamil Paruch, Lumír Krejčí, Aleš Hampl, Josef Jaroš, Milan Ešner, Radek Fedr, Peter Kollár, Petra Ovesná, Miroslava Bittová, Lucia Binó, Hana Kolářová, Kateřina Vašíčková, Petr Vaňhara, Nicol Straková, Stanislav Drápela, Fedor Nikulenkov, Prashant Khirsariya, Ondřej Hylse, Tereza Suchánková, Pounami Samadder
Detail description of Material and Methods
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ca2af450a9710cf2c3505b94fce584e3
https://doi.org/10.1158/1535-7163.22508937
https://doi.org/10.1158/1535-7163.22508937
Autor:
Karel Souček, Kamil Paruch, Lumír Krejčí, Aleš Hampl, Josef Jaroš, Milan Ešner, Radek Fedr, Peter Kollár, Petra Ovesná, Miroslava Bittová, Lucia Binó, Hana Kolářová, Kateřina Vašíčková, Petr Vaňhara, Nicol Straková, Stanislav Drápela, Fedor Nikulenkov, Prashant Khirsariya, Ondřej Hylse, Tereza Suchánková, Pounami Samadder
Figure S1. In vitro profiling of SCH900776 and its selected analogs. Figure S2. Analogs of MU380 with the N-trifluoromethylpyrazol pharmacophore. Figure S3. Dot plots of flow cytometric analysis using γH2AX and Annexin V staining related to Figure
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0c6e17c5a7b71ad8e67d5f9f5705156b
https://doi.org/10.1158/1535-7163.22508946
https://doi.org/10.1158/1535-7163.22508946
Autor:
Karel Souček, Kamil Paruch, Lumír Krejčí, Aleš Hampl, Josef Jaroš, Milan Ešner, Radek Fedr, Peter Kollár, Petra Ovesná, Miroslava Bittová, Lucia Binó, Hana Kolářová, Kateřina Vašíčková, Petr Vaňhara, Nicol Straková, Stanislav Drápela, Fedor Nikulenkov, Prashant Khirsariya, Ondřej Hylse, Tereza Suchánková, Pounami Samadder
Supplementary Legends for Supplemetary Figure S1 - S11
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2b70d35fe48e8779e16a93830d0eae56
https://doi.org/10.1158/1535-7163.22508940
https://doi.org/10.1158/1535-7163.22508940
Autor:
Lumir Krejci, Mário Špírek, Fletcher E. Bain, Daniel Nemecek, Sarah R. Hengel, Jana Prochazkova, Liping Yu, Brandon G Koch, Maria Spies, Jarmila Mlcouskova, Barbora Stefanovie, Fedor Nikulenkov
Publikováno v:
Nucleic Acids Research
The proper repair of deleterious DNA lesions such as double strand breaks prevents genomic instability and carcinogenesis. In yeast, the Rad52 protein mediates DSB repair via homologous recombination. In mammalian cells, despite the presence of the R
Autor:
Lumir Krejci, Manlio Vinciguerra, Marguerite Marie Le Pannérer, Oriana Lo Re, Valerio Pazienza, Šárka Pospíšilová, Carlo Riccardi, Fedor Nikulenkov, Oxana Bereshchenko, A. Francis Stewart, Giovanni Li Volti, Cesarina Giallongo, Marcus Buschbeck, Tommaso Mazza, Libor Červinek, Jana Kotašková, Giuseppe A. Palumbo, Sara Flamini
Publikováno v:
Dipòsit Digital de Documents de la UAB
Universitat Autònoma de Barcelona
Clinical Epigenetics
Recercat: Dipósit de la Recerca de Catalunya
Varias* (Consorci de Biblioteques Universitáries de Catalunya, Centre de Serveis Científics i Acadèmics de Catalunya)
r-IGTP. Repositorio Institucional de Producción Científica del Instituto de Investigación Germans Trias i Pujol
instname
Recercat. Dipósit de la Recerca de Catalunya
Universitat Autònoma de Barcelona
Clinical Epigenetics
Recercat: Dipósit de la Recerca de Catalunya
Varias* (Consorci de Biblioteques Universitáries de Catalunya, Centre de Serveis Científics i Acadèmics de Catalunya)
r-IGTP. Repositorio Institucional de Producción Científica del Instituto de Investigación Germans Trias i Pujol
instname
Recercat. Dipósit de la Recerca de Catalunya
Background Epigenetic regulation is important in hematopoiesis, but the involvement of histone variants is poorly understood. Myelodysplastic syndromes (MDS) are heterogeneous clonal hematopoietic stem cell (HSC) disorders characterized by ineffectiv
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::04053792e76e338e702e059bbe66bddb
https://ddd.uab.cat/record/223380
https://ddd.uab.cat/record/223380
Autor:
Elisabeth Hedström, Alexander E. Kel, Fedor Nikulenkov, Natalia Issaeva, Galina Selivanova, H Li, Yao Shi, Razif Gabdoulline, Elias S.J. Arnér, Joanna Zawacka-Pankau, Jianqiang Xu, Sofi E. Eriksson
Publikováno v:
Cell Death and Differentiation; Vol 21
Cell Death and Differentiation
Cell Death and Differentiation
Rescue of the p53 tumor suppressor is an attractive cancer therapy approach. However, pharmacologically activated p53 can induce diverse responses ranging from cell death to growth arrest and DNA repair, which limits the efficient application of p53-
Autor:
Miroslav Chovanec, Danuša Vlasáková, Lumir Krejci, Zuzana Slezakova, Dana Vigasova, Rainer Gith, Dorothea Anrather, Prabha Sarangi, Xiaolan Zhao, Fedor Nikulenkov, Veronika Altmannova, Peter Kolesar
Publikováno v:
Nucleic Acids Research
Non-homologous end-joining (NHEJ) repairs DNA double-strand breaks by tethering and ligating the two DNA ends. The mechanisms regulating NHEJ efficiency and interplay between its components are not fully understood. Here, we identify and characterize