Cdk5 Is Essential for Amphetamine to Increase Dendritic Spine Density in Hippocampal Pyramidal Neurons

Autor: Soledad Ferreras, Guillermo Fernández, Víctor Danelon, María V. Pisano, Luján Masseroni, Christopher A. Chapleau, Favio A. Krapacher, Estela C. Mlewski, Daniel H. Mascó, Carlos Arias, Lucas Pozzo-Miller, María G. Paglini

Publikováno v: Frontiers in Cellular Neuroscience, Vol 11 (2017)

Psychostimulant drugs of abuse increase dendritic spine density in reward centers of the brain. However, little is known about their effects in the hippocampus, where activity-dependent changes in the density of dendritic spine are associated with le

Externí odkaz: https://doaj.org/article/43758b3653f7406dacbde7a6ed4e3117

Convergent dopamine and ALK4 signaling to PCBP1 controls FosB alternative splicing and cocaine behavioral sensitization

Autor: Favio A Krapacher, Diana Fernández‐Suárez, Annika Andersson, Alvaro Carrier‐Ruiz, Carlos F Ibáñez

Publikováno v: The EMBO journalReferences. 41(15)

ΔfosB is an alternatively spliced product of the FosB gene that is essential for dopamine-induced reward pathways and that acts as a master switch for addiction. However, the molecular mechanisms of its generation and regulation by dopamine signalin

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0e15d8e2b1a61edfe2fb69d82822d8e6
https://pubmed.ncbi.nlm.nih.gov/35730718

Lack of Cdk5 activity is involved on Dopamine Transporter expression and function: Evidences from an animal model of Attention-Deficit Hyperactivity Disorder

Autor: Antonella Montemerlo, Gonzalo Quassollo, Claudia Bregonzio, Favio Ariel Krapacher, Carlos Arias, María Gabriela Paglini, María Dolores Rubianes, Soledad Ferreras, Guillermo Fernández, María V. Pisano, Macarena Mariel Mari

Attention deficit/Hyperactivity disorder (ADHD) is one of the most diagnosed psychiatric disorders nowadays. The core symptoms of the condition include hyperactivity, impulsiveness and inattention. The main pharmacological treatment consists of psych

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::aa98e50fb76b1d6d7639cf6892b4e2d9
https://doi.org/10.1101/2021.03.19.436226

Correction: MAG induces apoptosis in cerebellar granule neurons through p75NTR demarcating granule layer/white matter boundary

Autor: Diana Fernández-Suárez, Favio A. Krapacher, Annika Andersson, Carlos F. Ibáñez, Lilian Kisiswa

Publikováno v: Cell Death & Disease. 10

An amendment to this paper has been published and can be accessed via a link at the top of the paper.

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::a527f853e22650e853dd8d6d22083269
https://doi.org/10.1038/s41419-019-2058-3

MAG induces apoptosis in cerebellar granule neurons through p75NTR demarcating granule layer/white matter boundary

Autor: Diana Fernández-Suárez, Favio A. Krapacher, Annika Andersson, Carlos F. Ibáñez, Lilian Kisiswa

Publikováno v: Cell Death & Disease
Fernández-Suárez, D, Krapacher, F A, Andersson, A, Ibáñez, C F & Kisiswa, L 2019, ' MAG induces apoptosis in cerebellar granule neurons through p75 NTR demarcating granule layer/white matter boundary ', Cell Death and Disease, vol. 10, no. 10, 732 . https://doi.org/10.1038/s41419-019-1970-x
Cell Death and Disease, Vol 10, Iss 10, Pp 1-15 (2019)

MAG (Myelin-associated glycoprotein) is a type I transmembrane glycoprotein expressed by Schwann cells and oligodendrocytes, that has been implicated in the control of axonal growth in many neuronal populations including cerebellar granule neurons (C

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::5b0bd66e6a5d8470cbe09e153d696e31
http://europepmc.org/articles/PMC6768859