Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Fatima Benmohamed"'
Autor:
Sophie Laget, Lucile Broncy, Katia Hormigos, Dalia M Dhingra, Fatima BenMohamed, Thierry Capiod, Magne Osteras, Laurent Farinelli, Stephen Jackson, Patrizia Paterlini-Bréchot
Publikováno v:
PLoS ONE, Vol 12, Iss 1, p e0169427 (2017)
Circulating Tumor Cells (CTC) and Circulating Tumor Microemboli (CTM) are Circulating Rare Cells (CRC) which herald tumor invasion and are expected to provide an opportunity to improve the management of cancer patients. An unsolved technical issue in
Externí odkaz:
https://doaj.org/article/80957206b7f846fca0c7d6e7d3f28876
Autor:
Fatima Benmohamed, Mathieu Medina, Yong-Zheng Wu, Sophia Maschalidi, Gregory Jouvion, Laurent Guillemot, Michel Chignard, Bénédicte Manoury, Lhousseine Touqui
Publikováno v:
PLoS ONE, Vol 9, Iss 3, p e90466 (2014)
Pseudomonas aeruginosa is an opportunistic pathogen involved in nosocomial infections. While a number of studies have demonstrated the roles of TLR2, TLR4 and TLR5 in host defense againt P. aeruginosa infection, the implication of TLR9 in this proces
Externí odkaz:
https://doaj.org/article/97c5d3b48cd94223afe848a0fcf94253
Autor:
Fatima BenMohamed, Ignacio Garcia-Verdugo, Mathieu Medina, Viviane Balloy, Michel Chignard, Reuben Ramphal, Lhousseine Touqui
Publikováno v:
PLoS ONE, Vol 7, Iss 8 (2012)
Externí odkaz:
https://doaj.org/article/740d5f83fa844af8a673e8008b100820
Publikováno v:
Molecular and Cellular Biochemistry. 335:47-51
We recently reported that corticosteroids increase the expression of the T-type channel Ca(v)3.1 through a transcriptional up-regulation of the Ca(v)3.1 encoding gene cacna1g. The nucleotide sequence analysis of cacna1g promoter revealed putative glu
Autor:
Gilles Charpigny, Fatima BenMohamed, Michel Chignard, Sonja Tattermusch, Lhousseine Touqui, Ignacio Garcia-Verdugo, Dominique Leduc, Mario Ollero
Publikováno v:
European Respiratory Journal
European Respiratory Journal, European Respiratory Society, 2012, 40 (3), pp.714-723. ⟨10.1183/09031936.00023111⟩
European Respiratory Journal, 2012, 40 (3), pp.714-723. ⟨10.1183/09031936.00023111⟩
European Respiratory Journal, European Respiratory Society, 2012, 40 (3), pp.714-723. ⟨10.1183/09031936.00023111⟩
European Respiratory Journal, 2012, 40 (3), pp.714-723. ⟨10.1183/09031936.00023111⟩
Eicosanoids are metabolites of arachidonic acid produced by cyclooxygenases (COXs) or lipoxygenases (LOXs). They mediate inflammation and mucus secretion in chronic pulmonary inflammatory diseases. The gel-forming mucin MUC5AC is over-expressed in th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3b20816601d1445318526d90ea8fca76
https://hal.archives-ouvertes.fr/hal-01004450
https://hal.archives-ouvertes.fr/hal-01004450
Autor:
Christo Christov, Gilles Mangiapan, Philippe Lang, Marie Matignon, Dominique Desvaux, Karine Dahan, Fatima BenMohamed, Djillali Sahali, Virginie Ory, Maud Kamal, Shao-yu Zhang, André Pawlak, Marina Candelier, Georges Guellaen, Vincent Audard, Pierre Ronco, Véronique Bernard, Xavier Decrouy
Publikováno v:
Science Signaling
Science Signaling, American Association for the Advancement of Science, 2010, 3 (122), pp.ra39-ra39. ⟨10.1126/scisignal.2000678⟩
Science Signaling, 2010, 3 (122), pp.ra39-ra39. ⟨10.1126/scisignal.2000678⟩
Science Signaling, 2010, 3 (122), pp.ra39. ⟨10.1126/scisignal.2000678⟩
Science Signaling, American Association for the Advancement of Science (AAAS), 2010, 3 (122), pp.ra39. ⟨10.1126/scisignal.2000678⟩
Science Signaling, American Association for the Advancement of Science, 2010, 3 (122), pp.ra39. ⟨10.1126/scisignal.2000678⟩
Science Signaling, American Association for the Advancement of Science, 2010, 3 (122), pp.ra39-ra39. ⟨10.1126/scisignal.2000678⟩
Science Signaling, 2010, 3 (122), pp.ra39-ra39. ⟨10.1126/scisignal.2000678⟩
Science Signaling, 2010, 3 (122), pp.ra39. ⟨10.1126/scisignal.2000678⟩
Science Signaling, American Association for the Advancement of Science (AAAS), 2010, 3 (122), pp.ra39. ⟨10.1126/scisignal.2000678⟩
Science Signaling, American Association for the Advancement of Science, 2010, 3 (122), pp.ra39. ⟨10.1126/scisignal.2000678⟩
Idiopathic nephrotic syndrome comprises several podocyte diseases of unknown origin, affecting the glomerular podocyte, which plays a key role in controlling the permeability of the kidney filter to proteins. It is characterized by the daily loss of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0958ddc767d1431ea931f5b7657f7760
https://hal.archives-ouvertes.fr/hal-03013526
https://hal.archives-ouvertes.fr/hal-03013526
Autor:
Philippe Lang, Maud Kamal, Georges Guellaen, Asta Valanciuté, Karine Dahan, Djillali Sahali, Fatima BenMohamed, André Pawlak, Marina Candelier
Publikováno v:
FEBS letters. 584(3)
In naive T cells, Lck exerts a negative control on the ERK/MAPK pathway. We show that c-mip (c-maf inducing protein) interacts with the p85 subunit of PI3 kinase and inactivates Lck, which results in Erk1/2 and p38 MAPK activation. This effect is not
Autor:
Jean-François Renaud, Elodie Gouadon, Yann Ruchon, Laurent Ferron, Fatima BenMohamed, Véronique Capuano
Publikováno v:
Molecular and Cellular Biochemistry. 320
The effect of the dexamethasone (Dex) on the regulation of the T-type Ca(2+) channel expressions was investigated in primary cultures of neonatal rat ventricular myocytes. We found that Dex (1 microM) increases the T-type Ca(2+) current (I(CaT)) asso
Autor:
Bénédicte Manoury, Sophia Maschalidi, Yongzheng Wu, Grégory Jouvion, Lhousseine Touqui, Laurent Guillemot, Mathieu Medina, Fatima BenMohamed, Michel Chignard
Publikováno v:
PLoS ONE
PLoS ONE, Public Library of Science, 2014, 9 (3), pp.e90466. ⟨10.1371/journal.pone.0090466⟩
PLoS ONE, 2014, 9 (3), pp.e90466. ⟨10.1371/journal.pone.0090466⟩
PLoS ONE, Vol 9, Iss 3, p e90466 (2014)
PLoS ONE, Public Library of Science, 2014, 9 (3), pp.e90466. ⟨10.1371/journal.pone.0090466⟩
PLoS ONE, 2014, 9 (3), pp.e90466. ⟨10.1371/journal.pone.0090466⟩
PLoS ONE, Vol 9, Iss 3, p e90466 (2014)
International audience; Pseudomonas aeruginosa is an opportunistic pathogen involved in nosocomial infections. While a number of studies have demonstrated the roles of TLR2, TLR4 and TLR5 in host defense againt P. aeruginosa infection, the implicatio