Zobrazeno 1 - 10
of 23
pro vyhledávání: '"Fatemeh Mirzamohammadi"'
Autor:
Jialiang S. Wang, Tushar Kamath, Courtney M. Mazur, Fatemeh Mirzamohammadi, Daniel Rotter, Hironori Hojo, Christian D. Castro, Nicha Tokavanich, Rushi Patel, Nicolas Govea, Tetsuya Enishi, Yunshu Wu, Janaina da Silva Martins, Michael Bruce, Daniel J. Brooks, Mary L. Bouxsein, Danielle Tokarz, Charles P. Lin, Abdul Abdul, Evan Z. Macosko, Melissa Fiscaletti, Craig F. Munns, Pearl Ryder, Maria Kost-Alimova, Patrick Byrne, Beth Cimini, Makoto Fujiwara, Henry M. Kronenberg, Marc N. Wein
Publikováno v:
Nature Communications, Vol 12, Iss 1, Pp 1-20 (2021)
The molecular circuitry that drives dendrite formation during osteocytogenesis remains poorly understood. Here the authors show that deletion of Sp7, a gene linked to rare and common skeletal disease, in mature osteoblasts and osteocytes causes sever
Externí odkaz:
https://doaj.org/article/cd91057df8104b6bb42af0b42b4a9105
Autor:
Fatemeh Mirzamohammadi, Anastasia Kozlova, Garyfallia Papaioannou, Elena Paltrinieri, Ugur M. Ayturk, Tatsuya Kobayashi
Publikováno v:
Nature Communications, Vol 9, Iss 1, Pp 1-10 (2018)
Feingold syndrome is a skeletal dysplasia caused by mutations in MYCN or MIR17HG, but it is not clear if these mutations lead to pathology via a common molecular mechanism. Here, the authors show that mutations in MIR17HG lead to upregulated TGF-β s
Externí odkaz:
https://doaj.org/article/cce31e82791243b5bd7fb58a95b1fffa
Autor:
Fatemeh Mirzamohammadi, Garyfallia Papaioannou, Jennifer B. Inloes, Erinn B. Rankin, Huafeng Xie, Ernestina Schipani, Stuart H. Orkin, Tatsuya Kobayashi
Publikováno v:
Nature Communications, Vol 7, Iss 1, Pp 1-10 (2016)
Eed is a polycomb repressive complex 2 component involved in stem cell lineage determination, but little is known about its role in lineage committed cells. Here the authors show that chondrocyte-specific Eed KO mice have skeletal growth defects rela
Externí odkaz:
https://doaj.org/article/5a75c493278c44bfa5a6dabee56bba1b
Autor:
Faranak Sharifi, Yahya Jaberi, Fatemeh Mirzamohammadi, Hamid Mirzamohammadi, Nouraddin Mousavinasab
Publikováno v:
Indian Journal of Endocrinology and Metabolism, Vol 17, Iss 5, Pp 899-905 (2013)
Aims: To detect the risk factors of diabetes mellitus (DM) and cardiovascular complications in subjects with impaired fasting glucose (IFG). Materials and Methods: One hundred and twenty three subjects with proved IFG in Zanjan Healthy Heart Study (2
Externí odkaz:
https://doaj.org/article/3adfd29ebbf84577b96ca23776004f36
Autor:
Fatemeh Mirzamohammadi, Ogonna N. Nnamani Silva, Rebecca K. Leaf, Kyle R. Eberlin, Ian L. Valerio
Publikováno v:
Seminars in Plastic Surgery. 37:057-072
This review aims to highlight the common pharmacological and nonpharmacological interventions utilized for thromboprophylaxis as well as flap salvage in microsurgery. A literature review was conducted in PubMed/National Center for Biotechnology Infor
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::b37bb1d087bb7304336027bd256ce20e
https://doi.org/10.1055/s-0042-1760381
https://doi.org/10.1055/s-0042-1760381
Autor:
Marc N. Wein, Rushi Patel, Hironori Hojo, Beth A. Cimini, Jialiang S. Wang, Tetsuya Enishi, Charles P. Lin, Daniel J. Brooks, Fatemeh Mirzamohammadi, Michael Bruce, Christian D. Castro, Maria Kost-Alimova, Courtney M. Mazur, Danielle Tokarz, Craig F Munns, Evan Z. Macosko, Patrick J. Byrne, Abdul Abdul, Nicolas Govea, Makoto Fujiwara, Tushar Kamath, Henry M. Kronenberg, Mary L. Bouxsein, Melissa Fiscaletti, Nicha Tokavanich, Pearl V. Ryder, Janaina S. Martins, Yunshu Wu, Daniel Rotter
Publikováno v:
Nature Communications
Nature Communications, Vol 12, Iss 1, Pp 1-20 (2021)
Nature Communications, Vol 12, Iss 1, Pp 1-20 (2021)
Some osteoblasts embed within bone matrix, change shape, and become dendrite-bearing osteocytes. The circuitry that drives dendrite formation during “osteocytogenesis” is poorly understood. Here we show that deletion of Sp7 in osteoblasts and ost
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a4463b4a5e1ad52ca9eddbd196462dbf
http://europepmc.org/articles/PMC8560803
http://europepmc.org/articles/PMC8560803
Autor:
Shay Tzur, Hiroshi I. Suzuki, Gen Nishimura, Ann Nordgren, David R. Eyre, Elin Marsk, Ugur M. Ayturk, MaryAnn Weis, Giedre Grigelioniene, Fulya Taylan, Zvi Borochowitz, Eva Horemuzova, Tatsuya Kobayashi, Matthew L. Warman, Anna Lindstrand, Gintautas Grigelionis, Magnus Nordenskjöld, Anna Hammarsjö, Phillip A. Sharp, Fatemeh Mirzamohammadi
Publikováno v:
Nature medicine
MicroRNAs (miRNAs) are post-transcriptional regulators of gene expression. Heterozygous loss-of-function point mutations of miRNA genes are associated with several human congenital disorders1-5, but neomorphic (gain-of-new-function) mutations in miRN
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b3495540ca9ac61fc8a1ff0844344790
https://doi.org/10.1038/s41591-019-0353-2
https://doi.org/10.1038/s41591-019-0353-2
Autor:
Hironori Hojo, Michael Bruce, Henry M. Kronenberg, Mary L. Bouxsein, Jialiang S. Wang, Tetsuya Enishi, Christian D. Castro, Nicolas Govea, Rushi Patel, Makoto Fujiwara, Tushar Kamath, Daniel J. Brooks, Fatemeh Mirzamohammadi, Charles P. Lin, Janaina S. Martins, Yunshu Wu, Danielle Tokarz, Melissa Fiscaletti, Abdul Abdul, Daniel Rotter, Craig F Munns, Marc N. Wein, Evan Z. Macosko
Osteocytes use an elaborate network of dendritic connections to control bone remodeling. Some osteoblasts embed within mineralized bone matrix, change shape, and become osteocytes. The molecular circuitry that drives dendrite formation during “oste
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::1dd9f331e592441effcece751b832ced
https://doi.org/10.1101/2021.03.22.436056
https://doi.org/10.1101/2021.03.22.436056
Autor:
Anastasia Kozlova, Fatemeh Mirzamohammadi, Garyfallia Papaioannou, Ugur M. Ayturk, Elena Paltrinieri, Tatsuya Kobayashi
Publikováno v:
Nature Communications, Vol 9, Iss 1, Pp 1-10 (2018)
Nature Communications
Nature Communications
Feingold syndrome is a skeletal dysplasia caused by loss-of-function mutations of either MYCN (type 1) or MIR17HG that encodes miR-17-92 microRNAs (type 2). Since miR-17-92 expression is transcriptionally regulated by MYC transcription factors, it ha
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::17b992c1fa809bed067d746c37197dd4
https://doi.org/10.1038/s41467-018-03788-7
https://doi.org/10.1038/s41467-018-03788-7
Autor:
Marc N. Wein, Garyfallia Papaioannou, Tatsuya Kobayashi, Shigeki Nishimori, Fatemeh Mirzamohammadi, Thomas S. Lisse
Publikováno v:
Journal of Bone and Mineral Research. 30:1044-1052
Growth plate chondrocytes go through multiple differentiation steps and eventually become hypertrophic chondrocytes. The parathyroid hormone (PTH)-related peptide (PTHrP) signaling pathway plays a central role in regulation of hypertrophic differenti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4a79757791b3fbd34f81ba295781092f
https://doi.org/10.1002/jbmr.2438
https://doi.org/10.1002/jbmr.2438