Zobrazeno 1 - 10
of 48
pro vyhledávání: '"Fanny Reichert"'
Publikováno v:
Journal of Neuroinflammation, Vol 20, Iss 1, Pp 1-16 (2023)
Abstract Background Myelin that surrounds axons breaks in trauma and disease; e.g., peripheral nerve and spinal cord injuries (PNI and SCI) and multiple sclerosis (MS). Resulting myelin debris hinders repair if not effectively scavenged by Schwann ce
Externí odkaz:
https://doaj.org/article/8190f5f82b3c41e6b063ace809f89824
Autor:
Fanny Reichert, Keren Zohar, Elyad Lezmi, Tsiona Eliyahu, Shlomo Rotshenker, Michal Linial, Marta Weinstock
Publikováno v:
Biomolecules, Vol 14, Iss 1, p 112 (2024)
Treatment of aging rats for 6 months with ladostigil (1 mg/kg/day) prevented a decline in recognition and spatial memory and suppressed the overexpression of gene-encoding pro-inflammatory cytokines, TNFα, IL1β, and IL6 in the brain and microglial
Externí odkaz:
https://doaj.org/article/5be1dec6262341e399ae7efbe914f3bf
Autor:
Gerard Elberg, Sigal Liraz-Zaltsman, Fanny Reichert, Takashi Matozaki, Michael Tal, Shlomo Rotshenker
Publikováno v:
Journal of Neuroinflammation, Vol 16, Iss 1, Pp 1-14 (2019)
Abstract Background Recovery of function from traumatic nerve injury depends on the ability of severed axons to grow/regenerate back to their target tissues. This is achieved by successfully crossing the lesion site where physical impact severed axon
Externí odkaz:
https://doaj.org/article/69614fe77c6a4fc6a55d5baed287356e
Autor:
Fanny Reichert, Shlomo Rotshenker
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 13 (2019)
Myelin surrounding central nervous system (CNS) axons breaks down in multiple sclerosis (MS) and following traumatic axonal injury. Myelin-debris so produced is harmful to repair since it impedes remyelination in MS and the regeneration of traumatize
Externí odkaz:
https://doaj.org/article/ddc1df3bcf5a4b7b8b900fc2521c4f9f
Autor:
Chen Makranz, Goni Cohen, Ayellet Baron, Lital Levidor, Tatsuhiko Kodama, Fanny Reichert, Shlomo Rotshenker
Publikováno v:
Neurobiology of Disease, Vol 15, Iss 2, Pp 279-286 (2004)
Complement-receptor-3 (CR3/MAC-1), scavenger-receptor-AI/II (SRAI/II) and Fcγ-receptor (FcγR) can mediate phagocytosis of degenerated myelin in macrophages and microglia. However, CR3/MAC-1 and SRAI/II, but not FcγR, mediate phagocytosis after axo
Externí odkaz:
https://doaj.org/article/894aacce9cb84c1da285a34ba23e54c6
Autor:
Fanny Reichert, Shlomo Rotshenker
Publikováno v:
Neurobiology of Disease, Vol 12, Iss 1, Pp 65-72 (2003)
Microglia and macrophages express the αM/β2 integrin complement-receptor-3 (CR3/MAC-1; CD11b/CD18) and scavenger-receptor-AI/II (SRAI/II). Both can mediate myelin phagocytosis. We document that CR3/MAC-1 mediated myelin phagocytosis in microglia is
Externí odkaz:
https://doaj.org/article/e26de1144442422da66679a307a732e5
Publikováno v:
Neurobiology of Disease, Vol 8, Iss 3, Pp 504-512 (2001)
The removal of damaged myelin is central to repair after injury to axons and in autoimmune demyelinating diseases. Complement receptor 3 (CR3/MAC-1) plays a major role in mediating the phagocytosis of damaged myelin by macrophages and microglia. We s
Externí odkaz:
https://doaj.org/article/33585bba578d492481eaa8bc43bca2c3
Autor:
Chen Makranz, Goni Cohen, Ayellet Baron, Lital Levidor, Tatsuhiko Kodama, Fanny Reichert, Shlomo Rotshenker
Publikováno v:
Neurobiology of Disease, Vol 16, Iss 3, Pp 659- (2004)
Externí odkaz:
https://doaj.org/article/9d704eaa364c4d428f18180927c0c4e2
Autor:
Linial, Keren Zohar, Elyad Lezmi, Fanny Reichert, Tsiona Eliyahu, Shlomo Rotshenker, Marta Weinstock, Michal
Publikováno v:
International Journal of Molecular Sciences; Volume 24; Issue 13; Pages: 10928
The primary role of microglia is to maintain homeostasis by effectively responding to various disturbances. Activation of transcriptional programs determines the microglia’s response to external stimuli. In this study, we stimulated murine neonatal
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=multidiscipl::a69caf7d3b3b2570171fe844bf5c386d
Autor:
Michal Linial, Keren Zohar, Elyad Lezmi, Fanny Reichert, Tsiona Eliyahu, Shlomo Rotshenker, Marta Weinstock-Rosin
The primary role of microglia is to maintain homeostasis by effectively responding to various disturbances. Activation of transcriptional programs determines the microglia's response to external stimuli. In this study, we stimulated murine neonatal m
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f82b5d54827e311b4583fa3083cc8ffc
https://doi.org/10.21203/rs.3.rs-2724995/v1
https://doi.org/10.21203/rs.3.rs-2724995/v1
