Zobrazeno 1 - 10
of 31
pro vyhledávání: '"Fanhua Wei"'
Publikováno v:
Virulence, Vol 15, Iss 1 (2024)
Influenza A virus (IAV) is the leading cause of highly contagious respiratory infections, which poses a serious threat to public health. The non-structural protein 1 (NS1) is encoded by segment 8 of IAV genome and is expressed in high levels in host
Externí odkaz:
https://doaj.org/article/0631c75ac1e04ad9b882a67c4d8cc5f6
Publikováno v:
Shipin gongye ke-ji, Vol 44, Iss 2, Pp 293-298 (2023)
Objective: Exploring the disinfection effect of high-pressure carbon dioxide (HPCD) on human coronavirus 229E (hCoV-229E). Methods: The human coronavirus 229E (hCoV-229E) was treated by HPCD at different temperatures (10, 25, and 37 ℃) and pressure
Externí odkaz:
https://doaj.org/article/e2fe9788f2594ed29ce4c79094f15243
Publikováno v:
Frontiers in Microbiology, Vol 14 (2023)
Influenza viruses are one of the major causes of human respiratory infections and the newly emerging and re-emerging strains of influenza virus are the cause of seasonal epidemics and occasional pandemics, resulting in a huge threat to global public
Externí odkaz:
https://doaj.org/article/68f0b61f6e914f2697ed9e7069cf0db2
Publikováno v:
Frontiers in Microbiology, Vol 13 (2023)
Progranulin (PGRN) plays an important role in influenza virus infection. To gain insight into the potential molecular mechanisms by which PGRN regulates influenza viral replication, proteomic analyzes of whole mouse lung tissue from wild-type (WT) ve
Externí odkaz:
https://doaj.org/article/581a1fe156ca4c3cb1e5d7273f8d4c5d
Autor:
Fanhua Wei, Zhimin Jiang, Honglei Sun, Juan Pu, Yipeng Sun, Mingyang Wang, Qi Tong, Yuhai Bi, Xiaojing Ma, George Fu Gao, Jinhua Liu
Publikováno v:
PLoS Pathogens, Vol 16, Iss 2, p e1008321 (2020)
[This corrects the article DOI: 10.1371/journal.ppat.1008062.].
Externí odkaz:
https://doaj.org/article/0360153deaf640e0810f22bb77b91bd2
Autor:
Fanhua Wei, Zhimin Jiang, Honglei Sun, Juan Pu, Yipeng Sun, Mingyang Wang, Qi Tong, Yuhai Bi, Xiaojing Ma, George Fu Gao, Jinhua Liu
Publikováno v:
PLoS Pathogens, Vol 15, Iss 10, p e1008062 (2019)
Type I interferons (IFNs) play a critical role in host defense against influenza virus infection, and the mechanism of influenza virus to evade type I IFNs responses remains to be fully understood. Here, we found that progranulin (PGRN) was significa
Externí odkaz:
https://doaj.org/article/b5c7e6a8ef7d472893deaa75b398674f
Publikováno v:
Viruses, Vol 12, Iss 8, p 862 (2020)
Avian influenza A viruses (AIVs), as a zoonotic agent, dramatically impacts public health and the poultry industry. Although low pathogenic avian influenza virus (LPAIV) incidence and mortality are relatively low, the infected hosts can act as a viru
Externí odkaz:
https://doaj.org/article/bbb6a52dbb064657b6c0e54feef4e9d9
Publikováno v:
Mediators of Inflammation, Vol 2015 (2015)
The ADAMTS proteinases are a group of multidomain and secreted metalloproteinases containing the thrombospondin motifs. ADAMTS-7 is a member of ADAMTS family and plays a crucial role in the pathogenesis of arthritis. Overexpression of ADAMTS-7 gene p
Externí odkaz:
https://doaj.org/article/61f3ad5b279d43f0b23380498bd449a6
Progranulin facilitates conversion and function of regulatory T cells under inflammatory conditions.
Publikováno v:
PLoS ONE, Vol 9, Iss 11, p e112110 (2014)
The progranulin (PGRN) is known to protect regulatory T cells (Tregs) from a negative regulation by TNF-α, and its levels are elevated in various kinds of autoimmune diseases. Whether PGRN directly regulates the conversion of CD4+CD25-T cells into F
Externí odkaz:
https://doaj.org/article/731d2926afd148f291b4bc65420f55c0
Publikováno v:
Virology. 574
For influenza A viruses (IAVs), non-structural protein 1 (NS1) protein was recognized to be the key factor to enhance virulence by antagonizing host innate anti-viral responses. However, for the pathways allowing NS1 to regulate the type I interferon
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fe05a6208fecd4d1e99ceb276d7209e2
https://pubmed.ncbi.nlm.nih.gov/35926243
https://pubmed.ncbi.nlm.nih.gov/35926243