Zobrazeno 1 - 10
of 16
pro vyhledávání: '"Family Study Investigators"'
Autor:
Benedetta Izzi, Alessandro Gialluisi, Francesco Gianfagna, Sabatino Orlandi, Amalia De Curtis, Sara Magnacca, Simona Costanzo, Augusto Di Castelnuovo, Maria Benedetta Donati, Giovanni de Gaetano, Marc F. Hoylaerts, Chiara Cerletti, Licia Iacoviello, on behalf of the Moli-family Study Investigators
Publikováno v:
Cells, Vol 10, Iss 10, p 2737 (2021)
Defined as an index of platelet size heterogeneity, the platelet distribution width (PDW) is still a poorly characterized marker of platelet function in (sub)clinical disease. We presently validated PDW as a marker of P-selectin dependent platelet ac
Externí odkaz:
https://doaj.org/article/884491ad29cb4fe3b34e50b3f207e497
Autor:
Izzi, Benedetta, Gialluisi, Alessandro, Gianfagna, Francesco, Orlandi, Sabatino, Curtis, Amalia De, Magnacca, Sara, Costanzo, Simona, Castelnuovo, Augusto Di, Donati, Maria Benedetta, Gaetano, Giovanni de, Hoylaerts, Marc F., Cerletti, Chiara, Iacoviello, Licia, Investigators, on behalf of the Moli-family Study Investigators on behalf of the Moli-family Study
Publikováno v:
Cells
Volume 10
Issue 10
Cells, Vol 10, Iss 2737, p 2737 (2021)
Volume 10
Issue 10
Cells, Vol 10, Iss 2737, p 2737 (2021)
Defined as an index of platelet size heterogeneity, the platelet distribution width (PDW) is still a poorly characterized marker of platelet function in (sub)clinical disease. We presently validated PDW as a marker of P-selectin dependent platelet ac
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::49410eef18254ca109b3e0f19e537283
https://lirias.kuleuven.be/handle/123456789/684011
https://lirias.kuleuven.be/handle/123456789/684011
Autor:
Laskar, R, Ferreiro-Iglesias, A, Bishop, DT, Iles, MM, Kanetsky, PA, Armstrong, BK, Law, MH, Goldstein, AM, Aitken, JF, Giles, GG, Australian Melanoma Family Study Investigators, Leeds Case-Control Study Investigators, Robbins, HA, Cust, AE
Publikováno v:
The British journal of dermatology, vol 184, iss 6
BackgroundMelanoma aetiology has been proposed to have two pathways, which are determined by naevi and type of sun exposure and related to the anatomical site where melanoma develops.ObjectivesWe examined associations with melanoma by anatomical site
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::56e78a1ef14c49fa6ca923faf018c1b9
Autor:
Katherine S Elliott, Eleftheria Zeggini, Mark I McCarthy, Julius Gudmundsson, Patrick Sulem, Simon N Stacey, Steinunn Thorlacius, Laufey Amundadottir, Henrik Grönberg, Jianfeng Xu, Valerie Gaborieau, Rosalind A Eeles, David E Neal, Jenny L Donovan, Freddie C Hamdy, Kenneth Muir, Shih-Jen Hwang, Margaret R Spitz, Brent Zanke, Luis Carvajal-Carmona, Kevin M Brown, Australian Melanoma Family Study Investigators, Nicholas K Hayward, Stuart Macgregor, Ian P M Tomlinson, Mathieu Lemire, Christopher I Amos, Joanne M Murabito, William B Isaacs, Douglas F Easton, Paul Brennan, PanScan Consortium, Rosa B Barkardottir, Daniel F Gudbjartsson, Thorunn Rafnar, David J Hunter, Stephen J Chanock, Kari Stefansson, John P A Ioannidis
Publikováno v:
PLoS ONE, Vol 5, Iss 5, p e10858 (2010)
BackgroundGenome-wide association studies have found type 2 diabetes-associated variants in the HNF1B gene to exhibit reciprocal associations with prostate cancer risk. We aimed to identify whether these variants may have an effect on cancer risk in
Externí odkaz:
https://doaj.org/article/ed86bff323304eb0bb405d306b665368
Autor:
Cust, AE, Drummond, M, Kanetsky, PA, Australian Melanoma Family Study Investigators, Leeds case-control study Investigators, Goldstein, AM, Barrett, JH, MacGregor, S, Law, MH, Iles, MM, Bui, M, Hopper, JL, Brossard, M, Demenais, F, Taylor, JC, Hoggart, C, Brown, KM, Landi, MT, Newton-Bishop, JA, Mann, GJ, Bishop, DT
It is unclear to what degree genomic and traditional (phenotypic and environmental) risk factors overlap in their prediction of melanoma risk. We evaluated the incremental contribution of common genomic variants (in pigmentation, nevus and other path
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=core_ac_uk__::32d4eccfe13a72f75138465cfa98f98d
https://eprints.whiterose.ac.uk/131554/8/1-s2.0-S0022202X18320463-main.pdf
https://eprints.whiterose.ac.uk/131554/8/1-s2.0-S0022202X18320463-main.pdf
Autor:
Family Study Investigators, Pia Reece, Karleen M. Schulze, Michael M Cyr, Katherine M. Morrison, Sonia S. Anand, Stephanie A. Atkinson, Judah A. Denburg, Koon K. Teo, Tahira Batool
Publikováno v:
Journal of Developmental Origins of Health and Disease. 7:665-671
Prenatal and early-life environmental exposures play a key role in the development of atopy and allergic disease. The Family Atherosclerosis Monitoring In earLY life Study is a general, population-based Canadian birth cohort that prospectively evalua
Akademický článek
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Akademický článek
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Autor:
Anne E. Cust, Minh Bui, Australian Melanoma Family Study Investigators, Mark A. Jenkins, Chris Goumas
Publikováno v:
Cancer Epidemiology, Biomarkers & Prevention. 23:566-567
Genome-wide association studies have identified numerous common genomic variants associated with increased susceptibility to melanoma, but there is limited knowledge about the utility of adding them to risk prediction models for melanoma. Individuals
Autor:
Rosalind A. Eeles, Jenny L Donovan, Daniel F. Gudbjartsson, Douglas F. Easton, Rosa B. Barkardottir, Paul Brennan, Shih-Jen Hwang, Laufey T. Amundadottir, Brent W. Zanke, Australian Melanoma Family Study Investigators, Mark I. McCarthy, Mathieu Lemire, Ian Tomlinson, Thorunn Rafnar, Stuart MacGregor, Julius Gudmundsson, Kari Stefansson, Joanne M. Murabito, Freddie C. Hamdy, Steinunn Thorlacius, Patrick Sulem, Henrik Grönberg, Nicholas K. Hayward, Katherine S. Elliott, Eleftheria Zeggini, Christopher I. Amos, Stephen J. Chanock, David J. Hunter, Simon N. Stacey, William B. Isaacs, Jianfeng Xu, Kenneth Muir, David E. Neal, Kevin M. Brown, Margaret R. Spitz, Luis G. Carvajal-Carmona, John P. A. Ioannidis, Valerie Gaborieau
Publikováno v:
PloS one, vol 5, iss 5
PLoS ONE, Vol 5, Iss 5, p e10858 (2010)
PLoS ONE
PLoS ONE, Vol 5, Iss 5, p e10858 (2010)
PLoS ONE
Background\ud Genome-wide association studies have found type 2 diabetes-associated variants in the HNF1B gene to exhibit reciprocal associations with prostate cancer risk. We aimed to identify whether these variants may have an effect on cancer risk
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8dde547981a080c0281071ab5e157e08
https://escholarship.org/uc/item/08r1p719
https://escholarship.org/uc/item/08r1p719