Zobrazeno 1 - 10
of 848
pro vyhledávání: '"Fainsod, A."'
Autor:
Takaramoto, Shunki, Fainsod, Shai, Nagata, Takashi, Rozenberg, Andrey, Béjà, Oded, Inoue, Keiichi
Publikováno v:
In Journal of Molecular Biology 1 December 2024 436(23)
Publikováno v:
Ciencias Sociales y Educación, Vol 12, Iss 23, Pp 115-144 (2023)
El objetivo principal de este artículo es presentar los resultados parciales de la investigación realizada sobre la materia Ética (cátedra Cano/D’Iorio, FFyL, UBA), específicamente los modos en que allí se ponen en acción ciertos lineamiento
Externí odkaz:
https://doaj.org/article/4b1464d8044542c08b9aa24a3bc2bf27
Autor:
Leo McKay, Berardino Petrelli, Molly Pind, James N. Reynolds, Richard F. Wintle, Albert E. Chudley, Britt Drögemöller, Abraham Fainsod, Stephen W. Scherer, Ana Hanlon-Dearman, Geoffrey G. Hicks
Publikováno v:
Biomolecules, Vol 14, Iss 5, p 569 (2024)
Fetal Alcohol Spectrum Disorder (FASD) is a common neurodevelopmental disorder that affects an estimated 2–5% of North Americans. FASD is induced by prenatal alcohol exposure (PAE) during pregnancy and while there is a clear genetic contribution, f
Externí odkaz:
https://doaj.org/article/d3ecba3d3c3041e2b9255bc61b22cafc
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 11 (2023)
Introduction: Neural tube defects (NTDs) are among the most debilitating and common developmental defects in humans. The induction of NTDs has been attributed to abnormal folic acid (vitamin B9) metabolism, Wnt and BMP signaling, excess retinoic acid
Externí odkaz:
https://doaj.org/article/78671b6d61364fc29563005f04121777
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 11 (2023)
Fetal Alcohol Spectrum Disorder (FASD) arises from maternal consumption of alcohol during pregnancy affecting 2%–5% of the Western population. In Xenopus laevis studies, we showed that alcohol exposure during early gastrulation reduces retinoic aci
Externí odkaz:
https://doaj.org/article/73aa4d398f12401e89cb7425ea9409f8
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 10 (2022)
Retinoic acid (RA) is a central regulatory signal that controls numerous developmental processes in vertebrate embryos. Although activation of Hox expression is considered one of the earliest functions of RA signaling in the embryo, there is evidence
Externí odkaz:
https://doaj.org/article/5412626c186c4dc885df80098a40ab98
Publikováno v:
In Developmental Biology 15 June 2020 462(2):165-179
Autor:
Lishay Parhi, Tamar Alon-Maimon, Asaf Sol, Deborah Nejman, Amjad Shhadeh, Tanya Fainsod-Levi, Olga Yajuk, Batya Isaacson, Jawad Abed, Naseem Maalouf, Aviram Nissan, Judith Sandbank, Einav Yehuda-Shnaidman, Falk Ponath, Jörg Vogel, Ofer Mandelboim, Zvi Granot, Ravid Straussman, Gilad Bachrach
Publikováno v:
Nature Communications, Vol 11, Iss 1, Pp 1-12 (2020)
High levels of Fusobacterium nucleatum have been associated with poor overall survival in patients with colorectal and esophageal cancer. Here, the authors show that F. nucleatum is abundant in breast cancer samples and that the colonization by F. nu
Externí odkaz:
https://doaj.org/article/776f0ce45a884e0eb485ae59e2c301df
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 10 (2022)
Retinoic acid (RA) is a central signaling molecule regulating multiple developmental decisions during embryogenesis. Excess RA induces head malformations, primarily by expansion of posterior brain structures at the expense of anterior head regions, i
Externí odkaz:
https://doaj.org/article/198c67940d5d47398c1b6d4b9e4a456e
Autor:
Madhur Parihar, Liat Bendelac-Kapon, Michal Gur, Tali Abbou, Abha Belorkar, Sirisha Achanta, Keren Kinberg, Rajanikanth Vadigepalli, Abraham Fainsod
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 9 (2021)
Robustness is a feature of regulatory pathways to ensure signal consistency in light of environmental changes or genetic polymorphisms. The retinoic acid (RA) pathway, is a central developmental and tissue homeostasis regulatory signal, strongly depe
Externí odkaz:
https://doaj.org/article/f1794033bcd646a6a97139a9f24fb05a