Zobrazeno 1 - 10
of 35
pro vyhledávání: '"Fadia R, Homaidan"'
Publikováno v:
Mediators of Inflammation, Vol 12, Iss 5, Pp 277-283 (2003)
Background: Interleukin-1 (IL-1), an inflammatory cytokine whose levels are elevated in inflamed mucosa, causes part of its effect on intestinal epithelial cells (IEC) through inducing ceramide production.
Externí odkaz:
https://doaj.org/article/b1ad5b70354742daa7cfc3045d70b4ae
Publikováno v:
Mediators of Inflammation, Vol 11, Iss 1, Pp 39-45 (2002)
Background: In inflammatory bowel disease (IBD), cytokine levels (such as interleukin-1 (IL-1)) are elevated. We have shown previously that IL-1 activates phospholipid signaling pathways in intestinal epithelial cells (IEC), leading to increased cera
Externí odkaz:
https://doaj.org/article/c07ad54111f9496b99be7e7848d63229
Publikováno v:
Mediators of Inflammation, Vol 8, Iss 4-5, Pp 189-197 (1999)
Interleukin-1 (IL-1) is an inflammatory mediator that increases Cl- secretion in intestinal epithelial cells. To identify the signal transduction pathway(s) involved in IL-1's action, cells were treated with IL-1 and the levels of cyclooxygenase (COX
Externí odkaz:
https://doaj.org/article/8013da0051614affa713e2e545d274aa
Publikováno v:
Scientific Reports
Inflammatory bowel disease (IBD) involves functional impairment of intestinal epithelial cells (IECs), concomitant with the infiltration of the lamina propria by inflammatory cells. We explored the reciprocal paracrine and direct interaction between
The role of NF-κB in mediating the anti-inflammatory effects of IL-10 in intestinal epithelial cells
Publikováno v:
Cytokine. 36:1-8
In inflammatory bowel disease, cells that infiltrate the mucosa regulate intestinal epithelial cell function partly through release of pro- and anti-inflammatory cytokines. The aim of this study is to evaluate the role of the anti-inflammatory cytoki
Autor:
Imane Chakroun, Ghada Abou-Lteif, Asma Hatoum, Fadia R. Homaidan, Nadine Darwiche, Mohamed Kabbout
Publikováno v:
Molecular Carcinogenesis. 40:12-23
ASUMENDI A, 2002, BRIT J CANCER, V12, P1951; BOISE LH, 1993, CELL, V74, P597, DOI 10.1016-0092-8674(93)90508-N; Bruno S, 2002, CARCINOGENESIS, V23, P447, DOI 10.1093-carcin-23.3.447; Chambon P, 1996, FASEB J, V10, P940; Chan LNL, 1997, LEUKEMIA LYMPH
Publikováno v:
Mediators of Inflammation, Vol 11, Iss 1, Pp 39-45 (2002)
Mediators of Inflammation
Mediators of Inflammation
Background: In inflammatory bowel disease (IBD), cytokine levels (such as interleukin-1 (IL-1)) are elevated. We have shown previously that IL-1 activates phospholipid signaling pathways in intestinal epithelial cells (IEC), leading to increased cera
Publikováno v:
Inflammation Research. 50:375-381
Objective and Design: the aim of the study was to decipher the molecular signals involved in IL-1's action on intestinal epithelial cells (IEC).¶Materials and Methods: Mode-K cells, used as a model of IEC, were treated with IL-1, and PLA2 activity a
Publikováno v:
Inflammation. 23:191-205
A rabbit model of TNBS-colitis was used to study the effect of intestinal inflammation on epithelial cell function. Epithelial cells were isolated using a non-enzymatic isolation method without any apparent contamination with infiltrating immune cell
Publikováno v:
Digestive Diseases and Sciences. 44:1539-1546
This study was conducted to determine the iontransport mechanisms in the normal mouse cecum andcompare them to an inbred mouse model of colitis. TheUssing chamber-voltage clamp technique was used to monitor the short circuit current(Isc). The basal I