Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Fabrice Glibert"'
Autor:
Serguei, Nabirotchkin, Jan, Bouaziz, Fabrice, Glibert, Jonas, Mandel, Julie, Foucquier, Rodolphe, Hajj, Noëlle, Callizot, Nathalie, Cholet, Mickaël, Guedj, Daniel, Cohen
Publikováno v:
Journal of Alzheimer's Disease. 88:1585-1603
Background: Human diseases are multi-factorial biological phenomena resulting from perturbations of numerous functional networks. The complex nature of human diseases explains frequently observed marginal or transitory efficacy of mono-therapeutic in
Autor:
Yannick Pereira, Julien Laffaire, Gwenaël Primas, Emmanuel Vial, Jonas Mandel, Serguei Nabirotchkin, Aude Milet, Esther Graudens, Mickaël Guedj, Fabrice Glibert, Ilya Chumakov, Julie Foucquier, Michael W. Sereda, Daniel Cohen, Nathalie Cholet, Klaus-Armin Nave, Aurélie Boucard, Rodolphe Hajj, Viviane Bertrand
Publikováno v:
Orphanet Journal of Rare Diseases
Charcot-Marie-Tooth disease type 1A (CMT1A) is the most common inherited sensory and motor peripheral neuropathy. It is caused by PMP22 overexpression which leads to defects of peripheral myelination, loss of long axons, and progressive impairment th
Autor:
Laurent Susini, Stuart Pook, Bruno Lacroix, Isabelle Georges, Denis Le Paslier, Jennifer Lu-Kuo, David C. Ward, Isabelle Gros, Thomas Ried, Sandrine Beaufils, Christine Bellanné-Chantelot, Georges Lucotte, Alain Billault, Philippe Gesnouin, Ilya Chumakov, Daniel Cohen, Emmanuel Barillot, Fabrice Glibert, Guy Vaysseix, Stéphane Bertrand, Pierre Ougen, Jean-Jacques Codani
Publikováno v:
Cell. 70:1059-1068
Physical mapping of the human genome has until now been envisioned through single chromosome strategies. We demonstrate that by using large insert yeast artificial chromosomes (YACs) a whole genome approach becomes feasible. YACs (22,000) of 810 kb m
Autor:
Fabrice Glibert, Dung Le Nguyen, Odile Durieu-Trautmann, Pierre-Olivier Couraud, A. Donny Strosberg, Philippe Marin
Publikováno v:
European Journal of Pharmacology: Molecular Pharmacology. 206:191-198
Rat astrocytoma C6 cells have been recently identified as target cells for ET-1, which stimulates inositol lipid turnover in these cells. It is shown here that binding of ET-1 to high-affinity receptors on C6 cells leads to 40-45% inhibition of isopr