Zobrazeno 1 - 10
of 15
pro vyhledávání: '"Fabio Carrozzino"'
Autor:
Roberto Benelli, Fabio Carrozzino, Roberto Montesano, Manlio Vinciguerra, Marion Peyrou, Sebastiano Carlone, Michelangelo Foti
Publikováno v:
Journal of Hepatology, Vol. 50, No 6 (2009) pp. 1132-41
BACKGROUND/AIMS: The impact of dietary fatty acids on the development of cancers is highly controversial. We recently demonstrated that unsaturated fatty acids trigger the downregulation of the tumor suppressor PTEN through an mTOR/NF-kappaB-dependen
Autor:
Pascale Ribaux, Jan A. Ehses, Fabio Carrozzino, Nathalie Lin-Marq, Jean-Claude Irminger, Marianne Böni-Schnetzler, Philippe A. Halban, Marc Y. Donath, Eva Hammar
Publikováno v:
Endocrinology, Vol. 148, No 11 (2007) pp. 5582-5590
As we showed previously, the extracellular matrix (ECM) derived from rat bladder carcinoma cells (804G-ECM) has positive effects on rat primary beta-cell function and survival in vitro. The aim of this study was to define beta-cell genes induced by t
Publikováno v:
International Journal of Cancer. 85:21-26
Defective DNA repair has been suggested as a possible predisposing factor for breast cancer. We have investigated the repair of the frequent endogenous lesions abasic sites in sporadic early onset breast cancer patients and matched control individual
Autor:
Eleonora Gola, Walter Habeler, Stefano Indraccolo, Douglas M. Noonan, Luigi Chieco-Bianchi, Leonardo Santi, Alberto Amadori, Adriana Albini, Sonia Minuzzo, Fabio Carrozzino
Publikováno v:
Scopus-Elsevier
Antiangiogenic therapy may represent a promising approach to cancer treatment. Indeed, the efficacy of endogenous angiogenesis inhibitors, including angiostatin, endostatin and TIMPs, has been demonstrated in many types of solid tumors in animal mode
Publikováno v:
American Journal of Physiology. Cell Physiology, Vol. 297, No 3 (2009) pp. C775-87
Tight junctions (TJs) form a barrier to the paracellular diffusion of ions and solutes across epithelia. Although transmembrane proteins of the claudin family have emerged as critical determinants of TJ permeability, little is known about the signali
Publikováno v:
American Journal of Physiology-Renal Physiology, Vol. 296, No 2 (2009) pp. F446-57
Polycystic kidney diseases result from disruption of the genetically defined program that controls the size and geometry of renal tubules. Cysts which frequently arise from the collecting duct (CD) result from cell proliferation and fluid secretion.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::feb02765d5c52b5303625fc13544ccf0
https://archive-ouverte.unige.ch/unige:3876
https://archive-ouverte.unige.ch/unige:3876
Publikováno v:
BMC Developmental Biology, Vol. 7 (2007) P. 7
BMC Developmental Biology, Vol 7, Iss 1, p 7 (2007)
BMC Developmental Biology
BMC Developmental Biology, Vol 7, Iss 1, p 7 (2007)
BMC Developmental Biology
Background Formation of branching tubes is a fundamental step in the development of glandular organs. To identify extracellular cues that orchestrate epithelial tubulogenesis, we employed an in vitro assay in which EpH4-J3B1A mammary epithelial cells
Publikováno v:
Journal of Cell Science, Vol. 118, No Pt 15 (2005) pp. 3487-500
Although loss of cell-cell adhesion and gain of invasive properties play a crucial role in the malignant progression of epithelial tumours, the molecular signals that trigger these processes have not been fully elucidated. In light of the well-establ
Autor:
Amparo Cano, Noury Mensi, Fabio Carrozzino, Eric Féraille, Lelio Orci, Denise Huber, Priscilla Soulié, Roberto Montesano
Publikováno v:
American Journal of Physiology. Cell Physiology, Vol. 289, No 4 (2005) pp. C1002-14
Constitutive expression of the transcription factor Snail was previously shown to trigger complete epithelial-mesenchymal transition (EMT). The aim of this study was to determine whether inducible expression of Snail could modify epithelial propertie
Autor:
Alex Y. Strongin, Priscilla Soulié, Michael S. Pepper, Marie-France Poupon, Fabio Carrozzino, Roberto Montesano
Publikováno v:
Oncogene, Vol. 24, No 10 (2005) pp. 1689-97
Overexpression of membrane-type-1 matrix metalloproteinase (MT1-MMP) in tumor cells has previously been shown to enhance tumor growth and metastasis. To establish if MT1-MMP is also able to confer tumorigenicity on nonmalignant epithelial cells, we t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4d734b28f249642e4868a879bfb7313d
https://archive-ouverte.unige.ch/unige:3867
https://archive-ouverte.unige.ch/unige:3867