Zobrazeno 1 - 6
of 6
pro vyhledávání: '"FU‑QIN TANG"'
Publikováno v:
BMC Medical Informatics and Decision Making, Vol 24, Iss 1, Pp 1-17 (2024)
Abstract Background Heart failure is a syndrome with complex clinical manifestations. Due to increasing population aging, heart failure has become a major medical problem worldwide. In this study, we used the MIMIC-III public database to extract the
Externí odkaz:
https://doaj.org/article/74bb0c4595ea419581605a55dca8a553
Autor:
Fu-qin Tang, Zhao Jian, Yun Zhu, Yu Zhu, Yingbin Xiao, Hai-Long Wang, Wenjuan Yang, Ruiyan Ma, Qiong Zhu
Publikováno v:
Theranostics
Background: Oxygen supplementation in myocardial infarction (MI) remains controversial. Inflammation is widely believed to play a central role in myocardial repair. A better understanding of these processes may lead to the design of novel strategies
Autor:
Sai Chen, Zhao Jian, Yun‑Han Jiang, Shu‑Fan Zou, Yan Peng, Ying‑Bin Xiao, Fu‑Qin Tang, Lin Chen, Yang Yu
Publikováno v:
Molecular Medicine Reports.
The aim of the present study was to investigate the protective effect of Kruppel‑like factor 15 (KLF15) overexpression on heart failure (HF) induced by left ventricular (LV) pressure overload in mice. Wild‑type (WT) mice and cardiac‑specific KL
Autor:
Bo Liu, Hua‑Gang Zhang, Yun‑Han Jiang, Ying‑Bin Xiao, Fu‑Qin Tang, Gui‑Ping Luo, Zhao Jian, Yun Zhu
Publikováno v:
Molecular Medicine Reports
Induction of cardiomyocyte proliferation, the most promising approach to reverse myocardial attrition, has been gaining importance as a therapy for cardiovascular disease. Hypoxia and macrophages were previously independently reported to promote card
Autor:
Gui-Ping, Luo, Zhao, Jian, Rui-Yan, Ma, Zhe-Zhe, Cao, Yun, Zhu, Yu, Zhu, Fu-Qin, Tang, Ying-Bin, Xiao
Publikováno v:
International journal of clinical and experimental pathology. 11(12)
Hypoxia-induced apoptosis is an inevitable problem in cyanotic congenital heart disease. In the present study, we investigated effects of melatonin on hypoxic cardiomyocytes in vitro and in vivo, and explored its underlying mechanism. H9C2 cells were
Autor:
HUA‑GANG ZHANG, YUN ZHU, YUN‑HAN JIANG, GUI‑PING LUO, FU‑QIN TANG, ZHAO JIAN, YING‑BIN XIAO, BO LIU
Publikováno v:
Molecular Medicine Reports; Jun2017, Vol. 15 Issue 6, p3541-3548, 8p, 1 Chart, 4 Graphs