Kv7.4 channels regulate potassium permeability in neuronal mitochondria

Autor: Gianluca Paventi, Maria Virginia Soldovieri, Ilenio Servettini, Vincenzo Barrese, Francesco Miceli, Maria Josè Sisalli, Paolo Ambrosino, Ilaria Mosca, Iolanda Vinciguerra, Lara Testai, Antonella Scorziello, Gennaro Raimo, Vincenzo Calderone, Salvatore Passarella, Maurizio Taglialatela

Mitochondrial K+ permeability regulates neuronal apoptosis, energy metabolism, autophagy, and protection against ischemia–reperfusion injury. Kv7.4 channels have been recently shown to regulate K+ permeability in cardiac mitochondria and exert card

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::46d68172cd580b67b8431cd36679e1d7
http://hdl.handle.net/11588/873157

Activation of Kv7 potassium channels inhibits intracellular Ca2+ increases triggered by TRPV1-mediated pain-inducing stimuli in F11 immortalized sensory neurons

Autor: Maurizio Taglialatela, Francesco Miceli, Fabio Arturo Iannotti, Lorella M.T. Canzoniero, Ilaria Mosca, Maria Virginia Soldovieri, Erika Di Zazzo, Gianluca Paventi, Paolo Ambrosino, Cristina Franco

Publikováno v: International journal of molecular sciences (Online) 20 (2019). doi:10.3390/ijms20184322
info:cnr-pdr/source/autori:Ambrosino, Paolo; Soldovieri, Maria Virginia; Di Zazzo, Erika; Paventi, Gianluca; Iannotti, Fabio Arturo; Mosca, Ilaria; Miceli, Francesco; Franco, Cristina; Canzoniero, Lorella Maria Teresa; Taglialatela, Maurizio/titolo:Activation of Kv7 Potassium Channels Inhibits Intracellular Ca2+ Increases Triggered By TRPV1-Mediated Pain-Inducing Stimuli in F11 Immortalized Sensory Neurons/doi:10.3390%2Fijms20184322/rivista:International journal of molecular sciences (Online)/anno:2019/pagina_da:/pagina_a:/intervallo_pagine:/volume:20
International Journal of Molecular Sciences, Vol 20, Iss 18, p 4322 (2019)
International Journal of Molecular Sciences
Volume 20
Issue 18

Kv7.2-Kv7.5 channels mediate the M-current (IKM), a K+-selective current regulating neuronal excitability and representing an attractive target for pharmacological therapy against hyperexcitability diseases such as pain. Kv7 channels interact functio

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::08a746c76047ee7ab8abbd08c2bcd1ca
http://hdl.handle.net/11588/773272

Tento výsledek nelze pro nepřihlášené uživatele zobrazit.
K zobrazení výsledku je třeba se přihlásit.

Imidacloprid toxicity on cell culture and organism models

Autor: Malev, Olga, Fabbretti, Elsa, Trebše, Polonca

Modern agriculture has a great environmental impact and the ecological consequences of pesticide use are often of major concern. Pesticides are among the agricultural tools most popularly associated with environmental and human health harm. This rese

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=57a035e5b1ae::06a396f74e2150e720ca573f62943224
https://www.bib.irb.hr/823236

Effects of imidacloprid on F11 cellular model

Autor: Chen, Ke, Malev, Olga, Raspor, Lucija, Jun, Y, Trebše, Polonca, Fabbretti, Elsa

Neuronal excitability is a parameter related to neuron potential to generate signals of novel stimuli. Long-term exposure to environmental chemicals is believed to be a predisposing factor for further establishment of altered neuronal functions that

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=57a035e5b1ae::3a1d1216e5d6d487e027628debc6d6f5
https://www.bib.irb.hr/823224