Zobrazeno 1 - 5
of 5
pro vyhledávání: '"F. V. Pallardó"'
Autor:
M. Seco-Cervera, D. González-Rodríguez, J. S. Ibáñez-Cabellos, L. Peiró-Chova, P. González-Cabo, E. García-López, J. J. Vílchez, I. Sanz-Gallego, F. V. Pallardó, J. L. García-Giménez
Publikováno v:
Scientific Reports, Vol 7, Iss 1, Pp 1-12 (2017)
Abstract MicroRNAs (miRNAs) are noncoding RNAs that contribute to gene expression modulation by regulating important cellular pathways. In this study, we used small RNA sequencing to identify a series of circulating miRNAs in blood samples taken from
Externí odkaz:
https://doaj.org/article/e6d232ffeb4b41f99d56aff3acabb26b
Autor:
L, Gómez-Cambronero, B, Camps, J G, de La Asunción, M, Cerdá, A, Pellín, F V, Pallardó, J, Calvete, J H, Sweiry, G E, Mann, J, Viña, J, Sastre
Publikováno v:
The Journal of pharmacology and experimental therapeutics. 293(2)
Reactive oxygen radicals, nitric oxide, and cytokines have been implicated in the initiation of pancreatic tissue damage and impairment of the pancreatic microcirculation in acute pancreatitis. Pentoxifylline is a methylxanthine derivative with rheol
Autor:
J, Cortijo, M, Martí-Cabrera, J G, de la Asuncíon, F V, Pallardó, A, Esteras, L, Bruseghini, J, Viña, E J, Morcillo
Publikováno v:
Free radical biologymedicine. 27(3-4)
We examined the in vitro effects of tert-butylhydroperoxide (tBu-OOH) in human bronchial muscle. tert-Butylhydroperoxide produced concentration-dependent contractions of bronchial rings (maximum effect was 56.5 +/- 9.6% of contraction by 1 mM acetylc
Publikováno v:
Hepatology (Baltimore, Md.). 29(3)
Zidovudine (3'-azido-2',3'-dideoxythymidine [AZT]) inhibits human immunodeficiency virus replication and delays progression of acquired immune deficiency syndrome. We have recently found that, in muscle, AZT causes oxidative damage to mitochondrial D
Autor:
J, Sastre, F V, Pallardó, R, Plá, A, Pellín, G, Juan, J E, O'Connor, J M, Estrela, J, Miquel, J, Viña
Publikováno v:
Hepatology (Baltimore, Md.). 24(5)
Mitochondrial damage may be a major cause of cellular aging. So far, this hypothesis had only been tested using isolated mitochondria. The aim of this study was to investigate the involvement of mitochondria in aging using whole liver cells and not i