Zobrazeno 1 - 10
of 15
pro vyhledávání: '"F. Roeth"'
Publikováno v:
PLoS ONE, Vol 4, Iss 10, p e7634 (2009)
BACKGROUND:Tissue morphogenesis and organogenesis require that cells retain stable cell-cell adhesion while changing shape and moving. One mechanism to accommodate this plasticity in cell adhesion involves regulated trafficking of junctional proteins
Externí odkaz:
https://doaj.org/article/5a4c4d99a5af4435b318898798ae5149
Autor:
Malinda R Schaefer, Elizabeth R Wonderlich, Jeremiah F Roeth, Jolie A Leonard, Kathleen L Collins
Publikováno v:
PLoS Pathogens, Vol 4, Iss 8, p e1000131 (2008)
To facilitate viral infection and spread, HIV-1 Nef disrupts the surface expression of the viral receptor (CD4) and molecules capable of presenting HIV antigens to the immune system (MHC-I). To accomplish this, Nef binds to the cytoplasmic tails of b
Externí odkaz:
https://doaj.org/article/dbc005e6b5204967ae6130d072de254e
Autor:
J. Ferguson, George Thompson, Michele Curioni, S.J. Garcia-Vergara, F. Roeth, Teruo Hashimoto, Peter Skeldon
Publikováno v:
Surface and Interface Analysis. 42:234-240
Porous anodic oxides generated on copper-containing aluminium alloys are less regular than anodic oxides generated on pure aluminium. Specifically, a porous oxide morphology comprising layers of embryo pores, generated by a cyclic process of oxide fi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::addf32021e5102bfefb1373ab8e35678
https://doi.org/10.1002/sia.3139
https://doi.org/10.1002/sia.3139
Autor:
Kathleen L. Collins, Jeremiah F. Roeth
Publikováno v:
Microbiology and Molecular Biology Reviews. 70:548-563
SUMMARYThe Nef protein of primate lentiviruses is a unique protein that has evolved in several ways to manipulate the biology of an infected cell to support viral replication, immune evasion, pathogenesis, and viral spread. Nef is a small (25- to 34-
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0b729cc6abc0bc73bb860ba6294ac8dc
https://doi.org/10.1128/mmbr.00042-05
https://doi.org/10.1128/mmbr.00042-05
Autor:
Kathleen L. Collins, Rebekah I. Fleis, Matthew R. Kasper, Maya Williams, Tracey Filzen, Jeremiah F. Roeth
Publikováno v:
Journal of Biological Chemistry. 280:12840-12848
Human immunodeficiency virus, type 1 Nef disrupts viral antigen presentation and promotes viral immune evasion from cytotoxic T lymphocytes. There is evidence that Nef acts early in the secretory pathway to redirect major histocompatibility complex c
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::722c50ae330abee9f30987eecc2e952d
https://doi.org/10.1074/jbc.m413538200
https://doi.org/10.1074/jbc.m413538200
Publikováno v:
Journal of Virology. 79:632-636
Human immunodeficiency virus type 1 (HIV-1) Nef is a critical protein that is necessary for HIV pathogenesis. Its roles include the disruption of major histocompatibility complex class I (MHC-I) and CD4 trafficking to promote immune evasion and viral
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::58060db91999fa2d4217fa2e7b9b5093
https://doi.org/10.1128/jvi.79.1.632-636.2005
https://doi.org/10.1128/jvi.79.1.632-636.2005
Autor:
Jeremiah F. Roeth, Matthew R. Kasper, Kathleen L. Collins, Chris G. Przybycin, Rebekah I. Fleis, Maya Williams
Publikováno v:
Journal of Virology. 76:12173-12184
Nef, an essential pathogenic determinant for human immunodeficiency virus type 1, has multiple functions that include disruption of major histocompatibility complex class I molecules (MHC-I) and CD4 and CD28 cell surface expression. The effects of Ne
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2298bf729166c4bd1657bf175442b8d4
https://doi.org/10.1128/jvi.76.23.12173-12184.2002
https://doi.org/10.1128/jvi.76.23.12173-12184.2002
Publikováno v:
PLoS ONE, Vol 4, Iss 10, p e7634 (2009)
PLoS ONE
PLoS ONE
Background Tissue morphogenesis and organogenesis require that cells retain stable cell-cell adhesion while changing shape and moving. One mechanism to accommodate this plasticity in cell adhesion involves regulated trafficking of junctional proteins
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bef7ecbe5b40e9b8db2265d8d53cd8ba
http://europepmc.org/articles/PMC2763285?pdf=render
http://europepmc.org/articles/PMC2763285?pdf=render
Publikováno v:
The Journal of Cell Biology
To avoid immune recognition by cytotoxic T lymphocytes (CTLs), human immunodeficiency virus (HIV)-1 Nef disrupts the transport of major histocompatibility complex class I molecules (MHC-I) to the cell surface in HIV-infected T cells. However, the mec
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::07819ec80e19536d91c0f812a422f8de
https://pubmed.ncbi.nlm.nih.gov/15569716
https://pubmed.ncbi.nlm.nih.gov/15569716
Autor:
Elizabeth R. Wonderlich, Malinda Schaefer, Jeremiah F. Roeth, Kathleen L. Collins, Jolie A. Leonard
Publikováno v:
PLoS Pathogens
PLoS Pathogens, Vol 4, Iss 8, p e1000131 (2008)
PLoS Pathogens, Vol 4, Iss 8, p e1000131 (2008)
To facilitate viral infection and spread, HIV-1 Nef disrupts the surface expression of the viral receptor (CD4) and molecules capable of presenting HIV antigens to the immune system (MHC-I). To accomplish this, Nef binds to the cytoplasmic tails of b
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ee2a260d1efe6ed4fdd50f537e7e8bb9
https://doi.org/10.1371/journal.ppat.1000131
https://doi.org/10.1371/journal.ppat.1000131