Zobrazeno 1 - 4
of 4
pro vyhledávání: '"F. D. Kireev"'
Autor:
Yu. A. Lopatnikova, Yu. V. Zhukova, A. A. Alshevskaya, I. A. Obleukhova, F. D. Kireev, I. A. Belomestnova, S. V. Sennikov
Publikováno v:
Медицинская иммунология, Vol 25, Iss 3, Pp 459-464 (2023)
TNFa is a pro-inflammatory cytokine that is signaled through type 1 (TNFR1) and type 2 (TNFR2) receptors. TNFR1 normally mediates apoptosis, cell survival, and cytokine secretion, while TNFR2 selectively mediates cell survival and cytokine secretion.
Externí odkaz:
https://doaj.org/article/90c612f004394c449523be0cc6a339af
Autor:
A. A. Alshevskaya, J. V. Zhukova, J. A. Lopatnikova, F. D. Kireev, N. S. Shkaruba, O. A. Chumasova, J. A. Shevchenko, N. A. Ilina, A. E. Sizikov, S. V. Sennikov
Publikováno v:
Acta Biomedica Scientifica, Vol 7, Iss 5-1, Pp 154-166 (2022)
Background. The balance of TNF receptor expression on immune cells is a key factor determining cytokine-induced activation of proapoptotic or proliferative signaling pathways. As a result, the changes in cytokine level and in expression of its recept
Externí odkaz:
https://doaj.org/article/d7eeb1dcfce947a7b563a8cd06be6f4b
Autor:
Yu. V. Zhukova, A. A. Alshevskaya, F. D. Kireev, O. A. Chumasova, N. S. Shkaruba, A. E. Sizikov, Yu. A. Lopatnikova, D. V. Demina, A. V. Karaulov, A. N. Silkov, S. V. Sennikov
Publikováno v:
Медицинская иммунология, Vol 23, Iss 4, Pp 903-908 (2021)
A pleiotropic cytokine TNFα is an important inflammatory mediator of a number of diseases; its biological functions are fulfilled through two different receptors, TNFR1 and TNFR2. Changes in the ratio between these types of receptors shifting the ba
Externí odkaz:
https://doaj.org/article/482fd1a256c14ef8b21598e321ea6907
Autor:
F. D. Kireev, O. A. Chumasova, N. S. Shkaruba, Yu. V. Zhukova, Yu. A. Lopatnikova, A. N. Silkov, Sergey Sennikov, A. A. Alshevskaya, Aleksander V. Karaulov, A. Sizikov, D. V. Demina
Publikováno v:
Medicinskaâ Immunologiâ, Vol 23, Iss 4, Pp 903-908 (2021)
A pleiotropic cytokine TNFα is an important inflammatory mediator of a number of diseases; its biological functions are fulfilled through two different receptors, TNFR1 and TNFR2. Changes in the ratio between these types of receptors shifting the ba