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Autor:
J A, Roth, S G, Swisher, J A, Merritt, D D, Lawrence, B L, Kemp, C H, Carrasco, A K, El-Naggar, F V, Fossella, B S, Glisson, W K, Hong, F R, Khurl, J M, Kurie, J C, Nesbitt, K, Pisters, J B, Putnam, D S, Schrump, D M, Shin, G L, Walsh
Publikováno v:
Seminars in oncology. 25(3 Suppl 8)
The identification of genetic lesions that lead a normal cell to become malignant presents us with the opportunity of targeting those lesions as a means of therapy. Given the key role played by the tumor suppressor gene p53 in cell cycle regulation a