Zobrazeno 1 - 10
of 781
pro vyhledávání: '"F Malavasi"'
Autor:
S. Agarbati, D. Benfaremo, N. Viola, C. Paolini, S. Svegliati Baroni, A. Funaro, G. Moroncini, F. Malavasi, A. Gabrielli
Publikováno v:
Frontiers in Immunology, Vol 13 (2022)
ObjectiveCD38 is a type II glycoprotein highly expressed on plasmablasts and on short- and long-lived plasma cells, but weakly expressed by lymphoid, myeloid, and non-hematopoietic cells. CD38 is a target for therapies aimed at depleting antibody-pro
Externí odkaz:
https://doaj.org/article/77624dfee76a4bbf9bf521687c8879cc
Autor:
F. Morandi, I. Airoldi, A. Faini, A. Horenstein, F. Malavasi, N. Matysiak, K. Kopaczka, D. Marimpietri, R. Gramignoli
Publikováno v:
Human Immunology.
Autor:
F. Morandi, D. Marimpietri, A. L. Horenstein, M. Bolzoni, D. Toscani, F. Costa, B. Castella, A. C. Faini, M. Massaia, V. Pistoia, N. Giuliani, F. Malavasi
Publikováno v:
OncoImmunology, Vol 7, Iss 8 (2018)
Multiple myeloma (MM) derives from malignant transformation of plasma cells (PC), which accumulate in the bone marrow (BM), where microenvironment supports tumor growth and inhibits anti-tumor immune responses. Adenosine (ADO), an immunosuppressive m
Externí odkaz:
https://doaj.org/article/312cf86353544dc0ae3fb5ea398fcc1d
Publikováno v:
Mediators of Inflammation, Vol 2018 (2018)
Adenosine (ADO) is an immunosuppressive molecule, which suppresses the immune responses by interacting with specific receptors expressed by immune effector cells. ADO is produced from ATP through the enzymatic activities of CD39 and CD73. Alternative
Externí odkaz:
https://doaj.org/article/218dabc33e3147e5ad01916a7122e12e
Akademický článek
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Autor:
Silvia Sabbioni, Bahaeldin K. Elamin, Paola Guerriero, Elena Saccenti, Xiaomeng Huang, Massimo Negrini, George A. Calin, F. Malavasi, Antonella Bresin, Elisa Callegari, A. Chillemi, Ly James Lee, E. M.A.A. Hussein, Giorgio Zauli, Lucilla D'Abundo, Fabio Casciano, Paola Secchiero, Giandomenico Russo, Guido Marcucci, Carlo M. Croce
Publikováno v:
Oncogene. 36(47)
Dysregulation of microRNAs (miRNAs) plays an important role in the pathogenesis of chronic lymphocytic leukemia (CLL). The Eμ-TCL1 transgenic mouse develops a form of leukemia that is similar to the aggressive type of human B-CLL, and this valuable