Zobrazeno 1 - 10
of 115
pro vyhledávání: '"F K Conley"'
Publikováno v:
The Journal of Immunology. 153:4649-4654
Previous studies revealed that mice with the b or k allele at the H-2D region are susceptible to toxoplasmic encephalitis (TE); those with the d allele are resistant. To determine whether the b or d allele is dominant, F1 hybrids between susceptible
Publikováno v:
Infection and Immunity. 62:2773-2778
Treatment of toxoplasmic encephalitis in C57BL/6 mice with monoclonal antibody (MAb) against interleukin-6 (IL-6) resulted in a remarkable decrease in the number of foci of acute inflammation in their brains caused by proliferation of tachyzoites. In
Autor:
F K Conley
Publikováno v:
Radiology. 192:329-331
Publikováno v:
Infection and immunity. 61(6)
Resistance against acute and chronic infection with Taxoplasma gondii in BALB/c and CBA/Ca mice was compared. Intraperitoneal inoculation of either 20, 40, or 80 cysts of the ME49 strain resulted in mortality rates in BALB/c mice of 12% (2 of 17), 50
Publikováno v:
Immunology. 74(4)
Studies were performed in a murine model to determine if there is genetic control of the development of toxoplasmic encephalitis. Ten weeks after infection with the ME49 strain of Toxoplasma gondii, mice with the H-2b haplotype (C57BL/6, C57BL/10) an
Publikováno v:
AJNR Am J Neuroradiol
A murine model of implanted CNS neoplasia was used to study a new form of brain tumor immunotherapy with intralesional Corynebacterium parvum (C. parvum). Assessment of treatment protocols has been limited by the inability to assess, noninvasively, t
Autor:
F K, Conley
Publikováno v:
The Western journal of medicine. 153(5)
Publikováno v:
Journal of Kunming Medical University / Kunming Yike Daxue Xuebao. 2023, Vol. 44 Issue 12, p59-64. 6p.
Publikováno v:
Radiology. 192:325-325
Publikováno v:
The Journal of Immunology. 142:954-958
Toxoplasma gondii is an important cause of disease of the central nervous system in patients with AIDS. Among the variety of immunologic disorders encountered by AIDS patients is a depletion of CD4+ subpopulation of lymphocytes. In order to determine