Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Fábio Luis Forti"'
Autor:
José Luís Fachi, Jaqueline de Souza Felipe, Laís Passariello Pral, Bruna Karadi da Silva, Renan Oliveira Corrêa, Mirella Cristiny Pereira de Andrade, Denise Morais da Fonseca, Paulo José Basso, Niels Olsen Saraiva Câmara, Éricka Lorenna de Sales e Souza, Flaviano dos Santos Martins, Suzana Eiko Sato Guima, Andrew Maltez Thomas, João Carlos Setubal, Yuli Thamires Magalhães, Fábio Luis Forti, Thamiris Candreva, Hosana Gomes Rodrigues, Marcelo Bispo de Jesus, Sílvio Roberto Consonni, Alessandro dos Santos Farias, Patrick Varga-Weisz, Marco Aurélio Ramirez Vinolo
Publikováno v:
Cell Reports, Vol 27, Iss 3, Pp 750-761.e7 (2019)
Summary: Antibiotic-induced dysbiosis is a key factor predisposing intestinal infection by Clostridium difficile. Here, we show that interventions that restore butyrate intestinal levels mitigate clinical and pathological features of C. difficile-ind
Externí odkaz:
https://doaj.org/article/9c4fc043dfbe4f37bba8f567ccb37dd6
Publikováno v:
Cell Death and Disease, Vol 14, Iss 4, Pp 1-16 (2023)
Abstract Glioblastoma (GBM), the most common aggressive brain tumor, is characterized by rapid cellular infiltration and is routinely treated with ionizing radiation (IR), but therapeutic resistance inevitably recurs. The actin cytoskeleton of gliobl
Externí odkaz:
https://doaj.org/article/f6c621034ef349caa63da11eddd5fae8
Autor:
Lilian Cristina Russo, Jéssica Oliveira Farias, Pault Yeison Minaya Ferruzo, Lucas Falcão Monteiro, Fábio Luís Forti
Publikováno v:
Clinics, Vol 73, Iss suppl 1 (2018)
Protein tyrosine phosphatases have long been considered key regulators of biological processes and are therefore implicated in the origins of various human diseases. Heterozygosity, mutations, deletions, and the complete loss of some of these enzymes
Externí odkaz:
https://doaj.org/article/e37059fee7644f34acd6a1479cd11c13
Autor:
Fábio Luís Forti
Publikováno v:
Biblioteca Digital de Teses e Dissertações da USPUniversidade de São PauloUSP.
O hormônio adrenocorticotrópico, ACTH, regula função (esteroidogênese) e proliferação das células da córtex das glândulas adrenais através de um único receptor específico, ACTHR, que pertence à superfamília GPCR (G-protein coupled rece