Zobrazeno 1 - 10
of 22
pro vyhledávání: '"Ewa Dabrowska-Zamojcin"'
Autor:
Andrzej Pawlik, Maria Serdynska, Ewa Dabrowska-Zamojcin, Violetta Dziedziejko, Krzysztof Safranow, Leszek Domanski, Kazimierz Ciechanowski
Publikováno v:
Kidney & Blood Pressure Research, Vol 39, Iss 1, Pp 58-64 (2014)
Background/Aims: Renalase is a recently discovered protein, which is likely involved in regulation of blood pressure in humans and animals. Previous studies suggest that renalase reflects kidney functioning. A common missense polymorphism in the flav
Externí odkaz:
https://doaj.org/article/3675804eacea4c59ab7970dbec0e2a47
Publikováno v:
Journal of Clinical Medicine
Journal of Clinical Medicine, Vol 9, Iss 4045, p 4045 (2020)
Journal of Clinical Medicine, Vol 9, Iss 4045, p 4045 (2020)
Periodontal disease (PD) is a chronic inflammatory disease caused by the accumulation of bacterial plaque biofilm on the teeth and the host immune responses. PD pathogenesis is complex and includes genetic, environmental, and autoimmune factors. Nume
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ae00be103c5febc097b8c440933de1f5
http://europepmc.org/articles/PMC7765090
http://europepmc.org/articles/PMC7765090
Autor:
Andrzej Pawlik, Maciej Tarnowski, Krzysztof Safranow, Damian Malinowski, Michał Czerewaty, Sylwia Słuczanowska-Głabowska, Ewa Dabrowska-Zamojcin, Leszek Domański
Publikováno v:
Archivum Immunologiae et Therapiae Experimentalis
Ficolin-2 is an activator of the complement system that acts via the lectin pathway. Complement activation plays a substantial role in the renal injury inherent to kidney transplantation. In this study, we examined the associations between ficolin-2
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6be52246d9f73846f560187e20bc7c0e
http://europepmc.org/articles/PMC5767214
http://europepmc.org/articles/PMC5767214
Autor:
Krzysztof Safranow, Mateusz Kurzawski, Leszek Domański, Violetta Dziedziejko, Ewa Dabrowska-Zamojcin, Andrzej Pawlik
Publikováno v:
Transplant Immunology. 32:172-174
Background Fractalkine is a member of the chemokine family that acts as an adhesion molecule and as an extracellular chemoattractant promoting cellular migration. In this study, we analysed the association between the CX3CR1 gene V249I (rs3732379) SN
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0b51640a7b41098bb49f909cb586f35d
https://doi.org/10.1016/j.trim.2015.04.002
https://doi.org/10.1016/j.trim.2015.04.002
Publikováno v:
Pharmacogenomics. 15:905-907
2014 Understanding the pharmacogenetics of tacrolimus may help in adequate immunosuppression Evaluation of: Li CJ, Li L, Lin L et al. Impact of the CYP3A5, CYP3A4, COMT, IL-10 and POR genetic polymorphisms on tacrolimus metabolism in Chinese renal tr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::984b63a347354fb84aec82571f0bdb55
https://doi.org/10.2217/pgs.14.61
https://doi.org/10.2217/pgs.14.61
Autor:
Krzysztof Safranow, Andrzej Pawlik, Violetta Dziedziejko, Maciej Romanowski, Ewa Dabrowska-Zamojcin, Leszek Domański, Agnieszka Maciejewska-Karlowska, Marek Sawczuk
Publikováno v:
International immunopharmacology. 32
Post-transplant diabetes mellitus (PTDM) is a common complication after solid organ transplantation, especially in recipients treated with calcineurin inhibitors. Previous studies suggest that chronic inflammation and chemokines play an important rol
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f8715cdaebda80ce31dd8716077dc3aa
https://pubmed.ncbi.nlm.nih.gov/26802601
https://pubmed.ncbi.nlm.nih.gov/26802601
Publikováno v:
Annales Academiae Medicae Stetinensis. 59(2)
AIF-1 is an inflammatory cytokine influencing the immune system. There are increased levels of AIF-1 in the synovial fluid of patients with rheumatoid arthritis (RA). Sulfasalazine still remains the mainstay for the treatment of RA. It has an anti-in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::7f9f4edb69ba950682d004356a73a8e6
https://pubmed.ncbi.nlm.nih.gov/25026748
https://pubmed.ncbi.nlm.nih.gov/25026748
Autor:
Andrzej Pawlik, M Florczak, Barbara Gawrońska-Szklarz, Grażyna Dutkiewicz, Jacek Różański, Leszek Domański, Ewa Dabrowska-Zamojcin
Publikováno v:
Transplantation Proceedings. 37:2041-2043
Proinflammatory cytokines have been implicated in the pathogenesis of acute kidney allograft rejection. The aim of the study was to examine the association between interleukin (IL)-2 -330 and tumor necrosis factor (TNF)-alpha -308 promoter polymorphi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0d0c70a0ff82f90927f38aee373a599d
https://doi.org/10.1016/j.transproceed.2005.03.091
https://doi.org/10.1016/j.transproceed.2005.03.091
Autor:
Leszek Domański, Andrzej Pawlik, Ewa Dabrowska-Zamojcin, Violetta Dziedziejko, Krzysztof Safranow
Publikováno v:
Transplant immunology. 30(2-3)
CD28 is a costimulatory molecule which plays an important role in T cell-mediated immune response and transplantation. The aim of the present study was to examine the association between the IVS3 + 17T/C (rs3116496:T/C) polymorphism in the CD28 gene
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::98cf4e41db6c134c17e9adaf7bf484e1
https://pubmed.ncbi.nlm.nih.gov/24368148
https://pubmed.ncbi.nlm.nih.gov/24368148
Autor:
Andrzej, Pawlik, Leszek, Domanski, Jacek, Rozanski, Bogusław, Czerny, Zygmunt, Juzyszyn, Grazyna, Dutkiewicz, Marek, Myslak, Maciej, Hałasa, Marcin, Słojewski, Ewa, Dabrowska-Zamojcin
Publikováno v:
Annals of transplantation. 13(2)
Cytokines regulate cellular and humoral immune responses. One determinant of the variable cytokine production observed among individuals is genetic polymorphism in cytokine genes. These polymorphisms represent normal allelic variations in the cytokin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::9fbc92011a1c59cdb9cbbf17bcc1aaa4
https://pubmed.ncbi.nlm.nih.gov/18566561
https://pubmed.ncbi.nlm.nih.gov/18566561