Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Evelyn Nwabuisi-Heath"'
Autor:
Saverio Gentile, Erbo Dong, Monsheel S. Sodhi, Evelyn Nwabuisi-Heath, Alessandro Guidotti, Bristow Gc
BackgroundNeurodevelopmental deficits resulting from prenatal stress are associated with neurological disorders that include deficits of social behavior, such as schizophrenia1 and autism2–7. Studies of human brain and animal models indicate that a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::1afeeee63e8931f9af56ac3c62db7d03
https://doi.org/10.1101/2021.02.02.429408
https://doi.org/10.1101/2021.02.02.429408
Publikováno v:
ASN Neuro, Vol 6 (2013)
The ε4 allele of the gene that encodes apolipoprotein E (APOE4) is the greatest genetic risk factor for Alzheimer's disease (AD), while APOE2 reduces AD risk, compared to APOE3. The mechanism(s) underlying the effects of APOE on AD pathology remains
Externí odkaz:
https://doaj.org/article/3d8834b86bee4d26bc23eee898db068b
Publikováno v:
Journal of Neuroscience Methods. 207:137-147
Alterations in the density and morphology of dendritic spines are characteristic of multiple cognitive disorders. Elucidating the molecular mechanisms underlying spine alterations are facilitated by the use of experimental and analytical methods that
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0a94e2f041e95fa5133d32f3be894c9f
https://doi.org/10.1016/j.jneumeth.2012.04.003
https://doi.org/10.1016/j.jneumeth.2012.04.003
Autor:
Chunjiang Yu, Evelyn Nwabuisi-Heath, Sonya B. Dumanis, Yuan-Gui Zhu, G. William Rebeck, Leon M. Tai, Mary Jo LaDu
Publikováno v:
Glia. 60:559-569
The E4 allele of the Apolipoprotein E (APOE) gene is the strongest genetic risk factor for late-onset Alzheimer's disease (AD), and affects clinical outcomes of chronic and acute brain damages. The mechanisms by which apoE affect diverse diseases and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8067e64cffbe24f44f899f74a7036308
https://doi.org/10.1002/glia.22289
https://doi.org/10.1002/glia.22289
Publikováno v:
Biological Psychiatry. 81:S3
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::97b03a8d91dae42552de60cad9882106
https://doi.org/10.1016/j.biopsych.2017.02.017
https://doi.org/10.1016/j.biopsych.2017.02.017
Publikováno v:
ASN NEURO
ASN Neuro, Vol 6 (2013)
ASN Neuro, Vol 6 (2013)
The ε4 allele of the gene that encodes apolipoprotein E (APOE4) is the greatest genetic risk factor for Alzheimer's disease (AD), while APOE2 reduces AD risk, compared to APOE3. The mechanism(s) underlying the effects of APOE on AD pathology remains
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0c607b2ffd2f516dbc7faa205596c28e
https://pubmed.ncbi.nlm.nih.gov/24328732
https://pubmed.ncbi.nlm.nih.gov/24328732
Autor:
Lisa Jungbauer, Takahisa Kanekiyo, Chunjiang Yu, Mary Jo LaDu, Edwin J. Weeber, Jungsu Kim, G. William Rebeck, William A. Eimer, Steve Estus, Guojun Bu, Evelyn Nwabuisi-Heath, Ming Gan, Leon M. Tai, Katherine L. Youmans
APOE4 is the greatest risk factor for Alzheimer disease (AD) and synergistic effects with amyloid-β peptide (Aβ) suggest interactions among apoE isoforms and different forms of Aβ accumulation. However, it remains unclear how the APOE genotype aff
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::53bf9c4213f95f650d2d3b3749cbba22
https://europepmc.org/articles/PMC3516726/
https://europepmc.org/articles/PMC3516726/
Autor:
Sara Claude Michon, Takahisa Kanekiyo, William A. Eimer, Chunjiang Yu, Evelyn Nwabuisi-Heath, Sean M. Riordan, Katherine L. Youmans, Dean M. Hartley, Yifan Fu, Leon M. Tai, Arlene M. Manelli, Lester I. Binder, Guojun Bu, Mary Jo LaDu, W. Blaine Stine
Publikováno v:
Molecular Neurodegeneration, Vol 7, Iss 1, p 8 (2012)
Molecular Neurodegeneration
Molecular Neurodegeneration
Background The form(s) of amyloid-β peptide (Aβ) associated with the pathology characteristic of Alzheimer's disease (AD) remains unclear. In particular, the neurotoxicity of intraneuronal Aβ accumulation is an issue of considerable controversy; e
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::297a805b433a560dd15436a35f9cd27d
http://www.molecularneurodegeneration.com/content/7/1/8
http://www.molecularneurodegeneration.com/content/7/1/8
Publikováno v:
ASN Neuro (Sage Publications, Ltd.); 2014, Vol. 6 Issue 1, p21-28, 8p
Publikováno v:
Molecular Neurodegeneration. 2010, Vol. 5, p19-29. 11p.