Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Eva C. Dickinson"'
Autor:
Patricia Boyle, Recep Tuncer, Simon C. Watkins, Evan P. Nadler, Eva C. Dickinson, Sean Alber, Henri R. Ford
Publikováno v:
American Journal of Physiology-Gastrointestinal and Liver Physiology. 277:G1281-G1287
Endotoxemia promotes gut barrier failure and bacterial translocation (BT) by upregulating inducible nitric oxide synthase (iNOS) in the gut. We hypothesized that administration of a dithiocarbamate derivative, NOX, which scavenges nitric oxide (NO),
Autor:
Lynn Jensen, Eva C. Dickinson
Publikováno v:
The Journal of emergency medicine. 21(4)
Autor:
Eva C. Dickinson, Sean Alber, Evan P. Nadler, Simon C. Watkins, Donna Beer-Stolz, David W. Pratt, Henri R. Ford
Publikováno v:
American journal of physiology. Gastrointestinal and liver physiology. 281(1)
Sustained upregulation of inducible nitric oxide (NO) synthase in the liver after endotoxin [lipopolysaccharide (LPS)] challenge may result in hepatocellular injury. We hypothesized that administration of a NO scavenger, NOX, may attenuate LPS-induce
Autor:
Ugur Koltuksuz, Evan P. Nadler, Recep Tuncer, Simon C. Watkins, Patricia Boyle, Eva C. Dickinson, Sean Alber, Henri R. Ford
Publikováno v:
Journal of pediatric surgery. 35(7)
Background: Mucosal atrophy and bowel shortening are the hallmark of proximal intestinal diversion for extensive necrotizing enterocolitis (NEC) or Thiry-Vella fistulas (TVF), in which the ends of a defunctionalized loop of intestine are exteriorized
Autor:
Patricia Boyle, Henri R. Ford, Alex Knisely, Simon C. Watkins, Eva C. Dickinson, Donna Beer-Stolz, Evan P. Nadler, Xiao-Ru Zhang
Publikováno v:
The Journal of surgical research. 92(1)
Background. Previous investigators have relied on administration of pro-inflammatory cytokines or invasive surgical procedures to reproduce the morphologic changes of necrotizing enterocolitis (NEC) in rats. However, these artificial insults do not m
Publikováno v:
Seminars in pediatric surgery. 8(3)
The systemic inflammatory response syndrome (SIRS) is a leading cause of morbidity and mortality in adults and children. Various proinflammatory mediators have been implicated in the pathogenesis of SIRS; however, their mechanisms of action are poorl
Publikováno v:
The American Journal of Emergency Medicine. 20:62-63