Zobrazeno 1 - 10
of 132
pro vyhledávání: '"Eun-Jin Bae"'
Publikováno v:
Molecular Brain, Vol 16, Iss 1, Pp 1-4 (2023)
Abstract Parkinson’s disease (PD) is characterized by a selective loss of dopaminergic neurons. While most research on PD conducted to date has focused on neurons and, to a certain extent, glia, few studies have investigated changes in oligodendrog
Externí odkaz:
https://doaj.org/article/cefae0676bde45fd9e5eebcaae388fcb
Autor:
Eun-Jin Bae, Minsun Choi, Jeong Tae Kim, Dong-Kyu Kim, Min Kyo Jung, Changyoun Kim, Tae-Kyung Kim, Jun Sung Lee, Byung Chul Jung, Soo Jean Shin, Ka Hyun Rhee, Seung-Jae Lee
Publikováno v:
Experimental and Molecular Medicine, Vol 56, Iss 4, Pp 1027-1028 (2024)
Externí odkaz:
https://doaj.org/article/9248a607117e4f4b8bfed47f8b004b11
Autor:
Tae-Kyung Kim, Eun-Jin Bae, Byung Chul Jung, Minsun Choi, Soo Jean Shin, Sung Jun Park, Jeong Tae Kim, Min Kyo Jung, Ayse Ulusoy, Mi-Young Song, Jun Sung Lee, He-Jin Lee, Donato A. Di Monte, Seung-Jae Lee
Publikováno v:
Experimental and Molecular Medicine, Vol 56, Iss 4, Pp 1029-1030 (2024)
Externí odkaz:
https://doaj.org/article/50aba26acb94499fae6bd4b324b5db51
Autor:
Jihoon Lee, Ki Woon Sung, Eun-Jin Bae, Dabin Yoon, Dasarang Kim, Jin Saem Lee, Da-ha Park, Daniel Youngjae Park, Su Ran Mun, Soon Chul Kwon, Hye Yeon Kim, Joo-Ok Min, Seung-Jae Lee, Young Ho Suh, Yong Tae Kwon
Publikováno v:
Molecular Neurodegeneration, Vol 18, Iss 1, Pp 1-21 (2023)
Abstract Background There are currently no disease-modifying therapeutics for Parkinson’s disease (PD). Although extensive efforts were undertaken to develop therapeutic approaches to delay the symptoms of PD, untreated α-synuclein (α-syn) aggreg
Externí odkaz:
https://doaj.org/article/63f416f66800494bb59e84f43141648f
Autor:
Tae-Kyung Kim, Eun-Jin Bae, Byung Chul Jung, Minsun Choi, Soo Jean Shin, Sung Jun Park, Jeong Tae Kim, Min Kyo Jung, Ayse Ulusoy, Mi-Young Song, Jun Sung Lee, He-Jin Lee, Donato A. Di Monte, Seung-Jae Lee
Publikováno v:
Experimental and Molecular Medicine, Vol 54, Iss 12, Pp 2148-2161 (2022)
Abstract The clinical progression of neurodegenerative diseases correlates with the spread of proteinopathy in the brain. The current understanding of the mechanism of proteinopathy spread is far from complete. Here, we propose that inflammation is f
Externí odkaz:
https://doaj.org/article/99a88b201f584b40bda54e3a13392c78
Autor:
Eun-Jin Bae, Minsun Choi, Jeong Tae Kim, Dong-Kyu Kim, Min Kyo Jung, Changyoun Kim, Tae-Kyung Kim, Jun Sung Lee, Byung Chul Jung, Soo Jean Shin, Ka Hyun Rhee, Seung-Jae Lee
Publikováno v:
Experimental and Molecular Medicine, Vol 54, Iss 6, Pp 788-800 (2022)
Abstract Cell-to-cell propagation of α-synuclein is thought to be the underlying mechanism of Parkinson’s disease progression. Recent evidence suggests that inflammation plays an important role in the propagation of protein aggregates. However, th
Externí odkaz:
https://doaj.org/article/4731767c2b954ccca338bf9cebf0f22d
Autor:
Jihoon Lee, Ki Woon Sung, Eun-Jin Bae, Dabin Yoon, Dasarang Kim, Jin Saem Lee, Da-ha Park, Daniel Youngjae Park, Su Ran Mun, Soon Chul Kwon, Hye Yeon Kim, Joo-Ok Min, Seung-Jae Lee, Young Ho Suh, Yong Tae Kwon
Publikováno v:
Molecular Neurodegeneration, Vol 18, Iss 1, Pp 1-1 (2023)
Externí odkaz:
https://doaj.org/article/0f49e7ee9cb9412588f7f04d9182eeec
Autor:
Eun-Jin Bae, Dong-Kyu Kim, Changyoun Kim, Michael Mante, Anthony Adame, Edward Rockenstein, Ayse Ulusoy, Michael Klinkenberg, Ga Ram Jeong, Jae Ryul Bae, Cheolsoon Lee, He-Jin Lee, Byung-Dae Lee, Donato A. Di Monte, Eliezer Masliah, Seung-Jae Lee
Publikováno v:
Nature Communications, Vol 9, Iss 1, Pp 1-16 (2018)
Mutations in LRRK2 kinase are associated with Parkinson’s disease. Here the authors show that LRRK2 modulates propagation of α-synuclein, using rodent and C. elegans models, and show that this is dependent on phosphorylation of one of its substrat
Externí odkaz:
https://doaj.org/article/68a216617c8c47b59c9f66769ef9a5b0
Autor:
Se Hee Oh, Ha Na Kim, Hyun Jung Park, Jin Young Shin, Eun-Jin Bae, Mun Kyung Sunwoo, Seung-Jae Lee, Phil Hyu Lee
Publikováno v:
Cell Reports, Vol 14, Iss 4, Pp 835-849 (2016)
Ample evidence suggests that α-synuclein is released from cells and propagated from one area of the brain to others via cell-to-cell transmission. In terms of their prion-like behavior, α-synuclein propagation plays key roles in the pathogenesis an
Externí odkaz:
https://doaj.org/article/caf6147211164a75b2fd0dc357c87284
Autor:
Youngsang Ko, Eun Jin Bae, Sachin K Chitale, Carla Vieira Soares, Alexandre A. Leitão, Min-Kun Kim, Jong-San Chang, Guillaume Maurin, Sam Gon Ryu, U-Hwang Lee
Publikováno v:
ACS Applied Nano Materials. 5:9657-9665