Zobrazeno 1 - 10
of 56
pro vyhledávání: '"Eugene W. Krueger"'
Autor:
Omar L. Gutierrez-Ruiz, Katherine M. Johnson, Eugene W. Krueger, Roseanne E. Nooren, Nicole Cruz-Reyes, Carrie Jo Heppelmann, Tara L. Hogenson, Martin E. Fernandez-Zapico, Mark A. McNiven, Gina L. Razidlo
Publikováno v:
Cell Reports, Vol 42, Iss 9, Pp 113042- (2023)
Summary: Amplified lysosome activity is a hallmark of pancreatic ductal adenocarcinoma (PDAC) orchestrated by oncogenic KRAS that mediates tumor growth and metastasis, though the mechanisms underlying this phenomenon remain unclear. Using comparative
Externí odkaz:
https://doaj.org/article/9d68ce2bcbb5492f9eed1b1feddc32ac
Autor:
Masashi Ninomiya, Jun Inoue, Eugene W. Krueger, Jing Chen, Hong Cao, Atsushi Masamune, Mark A. McNiven
Publikováno v:
Hepatology Communications, Vol 5, Iss 7, Pp 1238-1251 (2021)
Currently, the hepatocellular trafficking pathways that are used by the hepatitis B virus (HBV) during viral infection and shedding are poorly defined. It is known that the HBV uses late endosomal and multivesicular body (MVB) compartments for assemb
Externí odkaz:
https://doaj.org/article/412340d7d6034a27bfd3e8616f084bbf
Autor:
Aneesha Dasgupta, Paige C. Arneson-Wissink, Rebecca E. Schmitt, Dong Seong Cho, Alexandra M. Ducharme, Tara L. Hogenson, Eugene W. Krueger, William R. Bamlet, Lizhi Zhang, Gina L. Razidlo, Martin E. Fernandez-Zapico, Jason D. Doles
Publikováno v:
JCI Insight, Vol 7, Iss 2 (2022)
Approximately 80% of pancreatic cancer patients suffer from cachexia, and one-third die due to cachexia-related complications such as respiratory failure and cardiac arrest. Although there has been considerable research into cachexia mechanisms and i
Externí odkaz:
https://doaj.org/article/fd3089a0330d47c2a75cc830a73c9afa
Autor:
Andrew P. Norgan, Jacqueline R. E. Lee, Andrea J. Oestreich, Johanna A. Payne, Eugene W. Krueger, David J. Katzmann
Publikováno v:
PLoS ONE, Vol 8, Iss 11 (2013)
Externí odkaz:
https://doaj.org/article/85a3dc3822c14e10b361e365c2365791
Supplementary Video 3 from A Novel Endocytic Mechanism of Epidermal Growth Factor Receptor Sequestration and Internalization
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7354dad6e380b1f26791580f69e442b9
https://doi.org/10.1158/0008-5472.22366659
https://doi.org/10.1158/0008-5472.22366659
Cells form transient, circular dorsal ruffles or “waves” in response to stimulation of receptor tyrosine kinases, including epidermal growth factor receptor (EGFR) or platelet-derived growth factor receptor. These dynamic structures progress inwa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7240cb3ca761cb1822fcbffa833b54f0
https://doi.org/10.1158/0008-5472.c.6495240
https://doi.org/10.1158/0008-5472.c.6495240
Supplementary Figure 3 from A Novel Endocytic Mechanism of Epidermal Growth Factor Receptor Sequestration and Internalization
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::60d97f051a8d286f3d77961e1bf99e4c
https://doi.org/10.1158/0008-5472.22366680.v1
https://doi.org/10.1158/0008-5472.22366680.v1
Supplementary Table 1 from A Novel Endocytic Mechanism of Epidermal Growth Factor Receptor Sequestration and Internalization
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a0178fafd189af078c066a7474621fee
https://doi.org/10.1158/0008-5472.22366674.v1
https://doi.org/10.1158/0008-5472.22366674.v1
Supplementary Figure and Video Legends 1-3 from A Novel Endocytic Mechanism of Epidermal Growth Factor Receptor Sequestration and Internalization
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1a7bc4d398a1c571216b72080dabc919
https://doi.org/10.1158/0008-5472.22366677
https://doi.org/10.1158/0008-5472.22366677
Supplementary Video 2 from A Novel Endocytic Mechanism of Epidermal Growth Factor Receptor Sequestration and Internalization
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::973db9d4134b0bc7c120269bd2818fb8
https://doi.org/10.1158/0008-5472.22366662.v1
https://doi.org/10.1158/0008-5472.22366662.v1