Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Etay Aloni"'
Publikováno v:
Frontiers in Neuroscience, Vol 17 (2023)
The medial amygdala (MeA) is a sexually dimorphic brain region that regulates fear responses, emotional memories, and social behaviors. It is known to be larger and contains more cells in males. The MeA integrates information through input connection
Externí odkaz:
https://doaj.org/article/26bfd23d538f4b46a6664191d61be0a9
Publikováno v:
eLife, Vol 12 (2023)
The mouse brain is by far the most intensively studied among mammalian brains, yet basic measures of its cytoarchitecture remain obscure. For example, quantifying cell numbers, and the interplay of sex, strain, and individual variability in cell dens
Externí odkaz:
https://doaj.org/article/8686c8b2da46415fb2ea238a0aea061b
Synaptopodin (SP) is localized within the spine apparatus, an enigmatic structure located in the neck of spines of central excitatory neurons. It serves as a link between the spine head, where the synapse is located, and the endoplasmic reticulum (ER
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::bb353a4385255c6358f02034fcb87fd6
https://doi.org/10.21203/rs.3.rs-494756/v1
https://doi.org/10.21203/rs.3.rs-494756/v1
Publikováno v:
Brain structurefunction. 226(7)
Synaptopodin (SP) is localized within the spine apparatus, an enigmatic structure located in the neck of spines of central excitatory neurons. It serves as a link between the spine head, where the synapse is located, and the endoplasmic reticulum (ER
Publikováno v:
The Journal of Neuroscience. 39:3983-3992
Disruption in calcium homeostasis is linked to several pathologies and is suggested to play a pivotal role in the cascade of events leading to Alzheimer's disease (AD). Synaptopodin (SP) residing in dendritic spines has been associated with ryanodine
Publikováno v:
Neuroscience. 394:156-163
Mitochondrial Carrier Homolog 2 (MTCH2) acts as a receptor for the BH3 interacting-domain death agonist (BID) in the mitochondrial outer membrane. Loss of MTCH2 affects mitochondria energy metabolism and function. MTCH2 forebrain conditional KO (MTCH
Autor:
Gemma E. White, Nicolas Palanca-Castan, Kouichi Maruyama, Eduardo Garza-Gisholt, Keiji Inohaya, Satz Mengensatzproduktion, Christine Köppl, Hagit Eldar-Finkelman, Akira Kudo, Masami Yoshimoto, Werner Druck Medien Ag, Tadashi Oishi, Shaun P. Collin, Culum Brown, Naoyuki Yamamoto, Moran Shapira, Nathan S. Hart, Hironobu Ito, Yuji Ishikawa, Etay Aloni, Ryan M. Kempster, John D. Pettigrew, Anat Barnea, Masayuki Iigo
Publikováno v:
Brain, Behavior and Evolution. 85:I-IV
Autor:
Eduard Korkotian, Efrat Oni-Biton, Ori Brenner, Atan Gross, Smadar Levin-Zaidman, Antonella Ruggiero, Yehudit Zaltsman, Michael Tsoory, Etay Aloni, Yael Kuperman, Liat Shachnai, Menahem Segal
Publikováno v:
Scientific Reports
Mitochondrial Carrier Homolog 2 (MTCH2) is a novel regulator of mitochondria metabolism, which was recently associated with Alzheimer’s disease. Here we demonstrate that deletion of forebrain MTCH2 increases mitochondria and whole-body energy metab