Inducible deletion of the prostaglandin EP3 receptor in kidney tubules of male and female mice has no major effect on water homeostasis.

Autor: Esteva-Font C; Department of Biomedicine, Aarhus University, Aarhus, Denmark., Geurts F; Division of Nephrology and Transplantation, Department of Internal Medicine, Erasmus Medical Center, University Medical Center Rotterdam, Rotterdam, The Netherlands., Hansen TPK; Department of Biomedicine, Aarhus University, Aarhus, Denmark., Hoorn EJ; Division of Nephrology and Transplantation, Department of Internal Medicine, Erasmus Medical Center, University Medical Center Rotterdam, Rotterdam, The Netherlands., Fenton RA; Department of Biomedicine, Aarhus University, Aarhus, Denmark.

Publikováno v: American journal of physiology. Renal physiology [Am J Physiol Renal Physiol] 2024 Sep 01; Vol. 327 (3), pp. F504-F518. Date of Electronic Publication: 2024 Jul 04.

Doxycycline Significantly Enhances Induction of Induced Pluripotent Stem Cells to Endoderm by Enhancing Survival Through Protein Kinase B Phosphorylation.

Autor: Peaslee C; Department of Pathology, University of California San Francisco, San Francisco, CA., Esteva-Font C; Department of Pathology, University of California San Francisco, San Francisco, CA., Su T; Department of Pathology, University of California San Francisco, San Francisco, CA., Munoz-Howell A; Children's Hospital Oakland Research Institute, University of California San Francisco, San Francisco, CA., Duwaerts CC; Department of Medicine, University of California San Francisco, San Francisco, CA.; Liver Center, University of California San Francisco, San Francisco, CA., Liu Z; Department of Cell and Tissue Biology, University of California San Francisco, San Francisco, CA.; Diabetes Center, University of California San Francisco, San Francisco, CA.; Eli and Edythe Broad Center for Regeneration Medicine and Stem Cell Research, University of California San Francisco, San Francisco, CA., Rao S; Department of Cell and Tissue Biology, University of California San Francisco, San Francisco, CA.; Diabetes Center, University of California San Francisco, San Francisco, CA.; Eli and Edythe Broad Center for Regeneration Medicine and Stem Cell Research, University of California San Francisco, San Francisco, CA., Liu K; Children's Hospital Oakland Research Institute, University of California San Francisco, San Francisco, CA., Medina M; Children's Hospital Oakland Research Institute, University of California San Francisco, San Francisco, CA.; Liver Center, University of California San Francisco, San Francisco, CA., Sneddon JB; Department of Cell and Tissue Biology, University of California San Francisco, San Francisco, CA.; Diabetes Center, University of California San Francisco, San Francisco, CA.; Eli and Edythe Broad Center for Regeneration Medicine and Stem Cell Research, University of California San Francisco, San Francisco, CA.; Department of Anatomy, University of California San Francisco, San Francisco, CA., Maher JJ; Department of Medicine, University of California San Francisco, San Francisco, CA.; Liver Center, University of California San Francisco, San Francisco, CA.; Eli and Edythe Broad Center for Regeneration Medicine and Stem Cell Research, University of California San Francisco, San Francisco, CA., Mattis AN; Department of Pathology, University of California San Francisco, San Francisco, CA.; Liver Center, University of California San Francisco, San Francisco, CA.

Publikováno v: Hepatology (Baltimore, Md.) [Hepatology] 2021 Oct; Vol. 74 (4), pp. 2102-2117.

Molecular biology of water and salt regulation in the kidney

Autor: Esteva-Font, C, Ballarin, J, Fernandez-Llama, P

Publikováno v: CELLULAR AND MOLECULAR LIFE SCIENCES
r-IIB SANT PAU. Repositorio Institucional de Producción Científica del Instituto de Investigación Biomédica Sant Pau
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The kidney plays a central role in the regulation of the salt and water balance, which depends upon an array of solute and water transporters in the renal tubules and upon vascular elements in the various regions of the kidney. Many recent studies ha

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=RECOLECTA___::437645a326d055fd48d16a67236f763c
https://iibsantpau.fundanetsuite.com/Publicaciones/ProdCientif/PublicacionFrw.aspx?id=11519

Rapid Aldosterone-Mediated Signaling in the DCT Increases Activity of the Thiazide-Sensitive NaCl Cotransporter.

Autor: Cheng L; InterPrET Center, Department of Biomedicine, Aarhus University, Aarhus, Denmark., Poulsen SB; InterPrET Center, Department of Biomedicine, Aarhus University, Aarhus, Denmark., Wu Q; InterPrET Center, Department of Biomedicine, Aarhus University, Aarhus, Denmark., Esteva-Font C; InterPrET Center, Department of Biomedicine, Aarhus University, Aarhus, Denmark., Olesen ETB; InterPrET Center, Department of Biomedicine, Aarhus University, Aarhus, Denmark.; Faculty of Health and Medical Sciences, Department of Biomedical Sciences, University of Copenhagen, Copenhagen, Denmark., Peng L; InterPrET Center, Department of Biomedicine, Aarhus University, Aarhus, Denmark., Olde B; Unit of Drug Target Discovery, Department of Experimental Medical Science, Lund University, Lund, Sweden., Leeb-Lundberg LMF; Unit of Drug Target Discovery, Department of Experimental Medical Science, Lund University, Lund, Sweden., Pisitkun T; InterPrET Center, Department of Biomedicine, Aarhus University, Aarhus, Denmark.; Faculty of Medicine, Chulalongkorn University, Bangkok, Thailand., Rieg T; Department of Molecular Pharmacology and Physiology, University of South Florida, Tampa, Florida., Dimke H; Department of Cardiovascular and Renal Research, Institute of Molecular Medicine, University of Southern Denmark, Odense, Denmark; and.; Department of Nephrology, Odense University Hospital, Odense, Denmark., Fenton RA; InterPrET Center, Department of Biomedicine, Aarhus University, Aarhus, Denmark; robert.a.fenton@biomed.au.dk.

Publikováno v: Journal of the American Society of Nephrology : JASN [J Am Soc Nephrol] 2019 Aug; Vol. 30 (8), pp. 1454-1470. Date of Electronic Publication: 2019 Jun 28.

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