Výsledky vyhledávání - "Estel Enreig"

Akademický článek

Glucocorticoids promote transition of ductal carcinoma in situ to invasive ductal carcinoma by inducing myoepithelial cell apoptosis

Autor: Arantzazu Zubeldia-Plazaola, Leire Recalde-Percaz, Núria Moragas, Mireia Alcaraz, Xieng Chen, Mario Mancino, Patricia Fernández-Nogueira, Miquel Prats de Puig, Flavia Guzman, Aleix Noguera-Castells, Anna López-Plana, Estel Enreig, Neus Carbó, Vanessa Almendro, Pedro Gascón, Paloma Bragado, Gemma Fuster

Publikováno v: Breast Cancer Research, Vol 20, Iss 1, Pp 1-16 (2018)

Abstract Background The microenvironment and stress factors like glucocorticoids have a strong influence on breast cancer progression but their role in the first stages of breast cancer and, particularly, in myoepithelial cell regulation remains uncl

Externí odkaz: https://doaj.org/article/09270c8358e54183a6b3aa8944386a95

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Supplementary Figure S2 from Tumor-Associated Fibroblasts Promote HER2-Targeted Therapy Resistance through FGFR2 Activation

Fibroblast effect on HER2 activation and HER2-targeted therapies sensitivity

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::09231ac70523ec6b34a3bc7388d36ca9
https://doi.org/10.1158/1078-0432.22473389

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Data from Tumor-Associated Fibroblasts Promote HER2-Targeted Therapy Resistance through FGFR2 Activation

Purpose:Despite the therapeutic success of existing HER2-targeted therapies, tumors invariably relapse. This study aimed at identifying new mechanisms responsible for HER2-targeted therapy resistance.Experimental Design:We have used a platform of HER

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::75503a5e3c93200695c9cee440396104
https://doi.org/10.1158/1078-0432.c.6528494

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Supplementary Legend from Tumor-Associated Fibroblasts Promote HER2-Targeted Therapy Resistance through FGFR2 Activation

Supplementary Legend

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::153a4e30a5aa46b033fbfa300e7a966b
https://doi.org/10.1158/1078-0432.22473374.v1

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Supplementary Material and methods from Tumor-Associated Fibroblasts Promote HER2-Targeted Therapy Resistance through FGFR2 Activation

File describing the supplementary material and methods and the table for the antibodies and drugs used for treatment

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::16ce7a3aa0167e9564d668793e69fb08
https://doi.org/10.1158/1078-0432.22473371.v1

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Tumor-Associated Fibroblasts Promote HER2-Targeted Therapy Resistance through FGFR2 Activation

Publikováno v: Clinical cancer research : an official journal of the American Association for Cancer Research
r-INCLIVA. Repositorio Institucional de Producción Científica de INCLIVA
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Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e227e565ba327bbbd44f395147ae2e89
https://www.fundanet.incliva.es/publicaciones/ProdCientif/PublicacionFrw.aspx?id=4333

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Histamine receptor 1 inhibition enhances antitumor therapeutic responses through extracellular signal-regulated kinase (ERK) activation in breast cancer

Autor: Patricia Jauregui, Núria Moragas, Paloma Bragado, Leire Recalde-Percaz, Aleix Noguera-Castells, Estel Enreig, Pedro Gascón, Vanessa Almendro, Mario Mancino, Patricia Fernández-Nogueira, Gemma Fuster, Anna López-Plana, Neus Carbó

Publikováno v: Cancer letters. 424

Histamine receptor 1 (HRH1) belongs to the rhodopsin-like G-protein-coupled receptor family. Its activation by histamine triggers cell proliferation, embryonic development, and tumor growth. We recently established that HRH1 is up-regulated in basal

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5b883177f3a53e9d6114d16b59c3e98e
https://pubmed.ncbi.nlm.nih.gov/29548821

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Abstract B69: Acquisition and maintenance of anti-Her2 target therapies resistance in breast cancer: Role of fibrobroblasts

Autor: Pere Gascón, Gemma Fuster, E. Ametller, Estel Enreig, Vanessa Almendro, Paloma Bragado, Mario Mancino

Publikováno v: Molecular Cancer Research. 14:B69-B69

Introduction: Mechanisms underlying tumor progression after chemotherapy are not well understood. Therapeutic treatments can favor the clonal selection of cells with unique properties and different fitness for a given microenvironment. Indeed, tumor

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::37f3ec75199bbf0a558b654aeb988767
https://doi.org/10.1158/1557-3125.advbc15-b69

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Abstract B100: Subtype specific expression of HRH1 contributes to increased chemoresistance of breast cancer cells

Autor: Mario Mancino, Elisabet Ametller, Pedro Gascón, Patricia Fernández-Nogueira, Vanessa Almendro, Estel Enreig

Publikováno v: Molecular Cancer Research. 11:B100-B100

Background: Histamine is a monoamine produced by α-decarboxylation of histidine by the enzyme histidine decarboxylase (HDC). Histamine acts as an autocrine regulator of cell proliferation through its binding to HRH1-4 receptors. In breast cancer (BC

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::7c9bf734c47fb62d737f84383c99f449
https://doi.org/10.1158/1557-3125.advbc-b100

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