Výsledky vyhledávání - "Espen Waage Skjeflo"

Quantification of Porcine Complement Activation Fragment C3a by a Neoepitope-Based Enzyme-Linked Immunosorbent Assay

Autor: Martin Oppermann, Kristin Pettersen, Hilde Fure, Espen Waage Skjeflo, Tom Eirik Mollnes, Dorte Christiansen, Per H. Nilsson

Publikováno v: The Complement System ISBN: 9781071610152

Enzyme-linked immunosorbent assay (ELISA) enables fast and simple quantification of analytes in the pico- to nanogram range in complex samples. Here, we describe an ELISA for the detection of porcine C3a as a marker for complement activation. Antibod

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::75a687ac0067eee6b6f36fb1983531ff
https://doi.org/10.1007/978-1-0716-1016-9_5

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Choice of immunoassay to evaluate porcine cytokine levels

Autor: Espen Waage Skjeflo, Tom Eirik Mollnes, Nora Lamark Ueland, Judith K. Ludvigsen, Bernt Christian Hellerud

Publikováno v: 230:110129
Veterinary Immunology and Immunopathology

Background In order to adequately monitor cytokines in experimental models, currently available methods and commercially available kits should be compared. Aim To compare the plasma and tissue concentrations of IL-1β, IL-6, IL-8, IL-10, and TNF as a

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2a492b4472b10058ecb932718bd11097
https://hdl.handle.net/10037/20921

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Phagocytosis of live and dead Escherichia coli and Staphylococcus aureus in human whole blood is markedly reduced by combined inhibition of C5aR1 and CD14

Autor: Anne Landsem, Terje Espevik, Dorte Christiansen, Jørgen Stenvik, Espen Waage Skjeflo, Tom Eirik Mollnes, Trent M. Woodruff, Erik Waage Nielsen

Publikováno v: Molecular Immunology

Source at https://doi.org/10.1016/j.molimm.2019.03.014. Background - Sepsis is a dysregulated host response to infection. The aim of this study was to investigate the effects of complement- and CD14 inhibition on phagocytosis of live and dead Gram-ne

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::64783421b8b0df80a2e5a8697559eef4
http://hdl.handle.net/11250/2632033

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Staphylococcus aureus-induced complement activation promotes tissue factor-mediated coagulation

Autor: Erik Waage Nielsen, Espen Waage Skjeflo, Trent M. Woodruff, Tom Eirik Mollnes, Dorte Christiansen, Terje Espevik, Judith K Ludviksen, Hilde Fure, Ole-Lars Brekke

Publikováno v: Journal of Thrombosis and Haemostasis

This is the peer reviewed version of the following article: Skjeflo, E.W., Christiansen, D., Fure, H., Ludviksen, J.K., Woodruff, T.M., Espevik, T., ... Mollnes, T.E. (2018). Staphylococcus aureus-induced complement activation promotes tissue factor-

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::768e43dbb26641eda94f424aa35368d2
http://hdl.handle.net/11250/2583362

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Combined Inhibition of Complement and CD14 Efficiently Attenuated the Inflammatory Response Induced by Staphylococcus aureus in a Human Whole Blood Model

Autor: Terje Espevik, Espen Waage Skjeflo, Tom Eirik Mollnes, Dorte Christiansen, Erik Waage Nielsen

Publikováno v: The Journal of Immunology. 192:2857-2864

The complement and TLR systems are activated in sepsis, contributing to an unfavorable inflammatory “storm.” Combined inhibition of these systems has been documented to efficiently attenuate the inflammatory responses induced by Gram-negative bac

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a618f8abe55d5bf0c2dc41bb194fcffa
https://doi.org/10.4049/jimmunol.1300755

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Combined inhibition of complement and CD14 improved outcome in porcine polymicrobial sepsis

Autor: Terje Espevik, Andreas Barratt-Due, Espen Waage Skjeflo, Ole-Lars Brekke, Caroline Sagatun, Knut Dybwik, Corinna Lau, Markus Huber-Lang, Hilde Fure, Tom Eirik Mollnes, Miles A. Nunn, Erik Waage Nielsen, Sven Haakon Urving, Sturla Aam

Publikováno v: Critical Care
19:415

Introduction Sepsis is an exaggerated and dysfunctional immune response to infection. Activation of innate immunity recognition systems including complement and the Toll-like receptor family initiate this disproportionate inflammatory response. The a

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7f32616cbe6615ec9549c11c919e6b0d
https://hdl.handle.net/11250/2380836

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