Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Erzsébet Bakk"'
Autor:
Laszlo Ducza, Andrea Gajtko, Krisztina Hegedűs, Erzsébet Bakk, Botond Gaál, Roland Takács, Péter Szücs, Krisztina Holló
Publikováno v:
IBRO Neuroscience Reports, Vol 15, Iss , Pp S678-S679 (2023)
Externí odkaz:
https://doaj.org/article/5a82515ad2484ceda7361ede7ad3a5c3
Autor:
László Ducza, Andrea Gajtkó, Krisztina Hegedűs, Erzsébet Bakk, Gréta Kis, Botond Gaál, Roland Takács, Péter Szücs, Klára Matesz, Krisztina Holló
Publikováno v:
Frontiers in Molecular Neuroscience, Vol 16 (2023)
ObjectiveIntense inflammation may result in pain, which manifests as spinal central sensitization. There is growing evidence that purinergic signaling plays a pivotal role in the orchestration of pain processing. Over the last decade the ionotropic P
Externí odkaz:
https://doaj.org/article/b375bf0966dd4d4f8fb3f83fe0c63d2b
Publikováno v:
Frontiers in Physiology, Vol 11 (2020)
It is now widely accepted that the glial cells of the central nervous system (CNS) are key players in many processes, especially when they are activated via neuron-glia or glia-glia interactions. In turn, many of the glia-derived pro-inflammatory cyt
Externí odkaz:
https://doaj.org/article/061f72718ea14657be7b75a51eb5a18c
Autor:
Krisztina Holló, László Ducza, Zoltán Hegyi, Klaudia Dócs, Krisztina Hegedűs, Erzsébet Bakk, Ildikó Papp, Gréta Kis, Zoltán Mészár, Zsuzsanna Bardóczi, Miklós Antal
Publikováno v:
Journal of Neuroinflammation, Vol 14, Iss 1, Pp 1-18 (2017)
Abstract Background All known biological functions of the pro-inflammatory cytokine interleukin-1β (IL-1β) are mediated by type 1 interleukin receptor (IL-1R1). IL-1β–IL-1R1 signaling modulates various neuronal functions including spinal pain pr
Externí odkaz:
https://doaj.org/article/5f71ac961e414590af363aec6a5d67fa
Autor:
Zoltán Mészár, Zoltán Hegyi, Ildikó Papp, Krisztina Hegedűs, Miklós Antal, László Ducza, Zsuzsanna Bardóczi, Krisztina Holló, Klaudia Dócs, Gréta Kis, Erzsébet Bakk
Publikováno v:
Journal of Neuroinflammation
Journal of Neuroinflammation, Vol 14, Iss 1, Pp 1-18 (2017)
Journal of Neuroinflammation, Vol 14, Iss 1, Pp 1-18 (2017)
Background All known biological functions of the pro-inflammatory cytokine interleukin-1β (IL-1β) are mediated by type 1 interleukin receptor (IL-1R1). IL-1β–IL-1R1 signaling modulates various neuronal functions including spinal pain processing.