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pro vyhledávání: '"Erik M. Edwards"'
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 290:H37-H45
ACh stimulates arachidonic acid (AA) release from membrane phospholipids of vascular endothelial cells (ECs). In rabbit aorta, AA is metabolized through the 15-lipoxygenase pathway to form vasodilatory eicosanoids 15-hydroxy-11,12-epoxyeicosatrienoic
Publikováno v:
Hypertension. 45:666-671
Bradykinin causes arterial relaxation and hyperpolarization, which is mediated by a transferable endothelium-derived hyperpolarizing factor (EDHF). In coronary arteries, epoxyeicosatrienoic acids (EETs) are involved in the EDHF response. However, the
Publikováno v:
Endocrinology. 146(8)
Adrenal steroidogenesis is modulated by humoral and neuronal factors and blood flow. Angiotensin II (AII) stimulates adrenal cortical aldosterone and cortisol production and medullary catecholamine release. However, AII regulation of adrenal vascular
Publikováno v:
Hypertension (Dallas, Tex. : 1979). 43(2)
Arachidonic acid induces an endothelium-dependent relaxation of the rabbit aorta that is blocked by lipoxygenase inhibitors. The cellular vasodilatory mechanisms activated by arachidonic acid metabolites remain undefined. In rabbit thoracic aortic ri
Publikováno v:
American Journal of Physiology: Heart & Circulatory Physiology; Jan2006, Vol. 290 Issue 1, pH37-H45, 9p, 1 Chart, 47 Graphs