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Autor:
Erik C Kuntzsch, Sarah T. Bjork, Joseph E. Mazurkiewicz, R. Liane Ramirez, Paul J. Feustel, Arnulf H. Koeppen
Publikováno v:
Acta Neuropathologica Communications
Background Friedreich ataxia (FA) causes distinctive lesions of dorsal root ganglia (DRG), including neuronal atrophy, satellite cell hyperplasia, and absorption of dying nerve cells into residual nodules. Two mechanisms may be involved: hypoplasia o