Zobrazeno 1 - 10
of 23
pro vyhledávání: '"Eric S Luth"'
Autor:
Eric S Luth, Molly Hodul, Bethany J Rennich, Carmino Riccio, Julia Hofer, Kaitlin Markoja, Peter Juo
Publikováno v:
PLoS Genetics, Vol 17, Iss 2, p e1009375 (2021)
Several intracellular trafficking pathways contribute to the regulation of AMPA receptor (AMPAR) levels at synapses and the control of synaptic strength. While much has been learned about these intracellular trafficking pathways, a major challenge is
Externí odkaz:
https://doaj.org/article/4892a406487643718c10c0b293203d00
Autor:
Lidia Park, Eric S Luth, Kelsey Jones, Julia Hofer, Irene Nguyen, Katherine E Watters, Peter Juo
Publikováno v:
PLoS ONE, Vol 16, Iss 2, p e0245587 (2021)
Regulation of AMPA-type glutamate receptor (AMPAR) expression and function alters synaptic strength and is a major mechanism underlying synaptic plasticity. Although transcription is required for some forms of synaptic plasticity, the transcription f
Externí odkaz:
https://doaj.org/article/5ff63ea762474fbd8d265ef8f6f4deb6
Publikováno v:
PLoS ONE, Vol 9, Iss 7, p e103727 (2014)
A switch in the conformational properties of α-synuclein (αS) is hypothesized to be a key step in the pathogenic mechanism of Parkinson's disease (PD). Whereas the beta-sheet-rich state of αS has long been associated with its pathological aggregat
Externí odkaz:
https://doaj.org/article/5f727051d4ae45da96724ad45cab150f
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 17 (2023)
AMPA receptors (AMPARs) mediate the majority of fast excitatory transmission in the brain. Regulation of AMPAR levels at synapses controls synaptic strength and underlies information storage and processing. Many proteins interact with the intracellul
Externí odkaz:
https://doaj.org/article/fbbc721e64e64c8a81da7d10b0d14b7d
Publikováno v:
J Neurosci
Reversible modification of AMPA receptors (AMPARs) with ubiquitin regulates receptor levels at synapses and controls synaptic strength. The conserved deubiquitinating enzyme (DUB) ubiquitin-specific protease-46 (USP-46) removes ubiquitin from AMPARs
Publikováno v:
PeerJ
PeerJ, Vol 9, p e10892 (2021)
PeerJ, Vol 9, p e10892 (2021)
The Doublesex/Mab-3 Domain transcription factor DMD-10 is expressed in several cell types in C. elegans, including in the nervous system. We sought to investigate whether DMD-10 is required for normal neuronal function using behavioral assays. We fou
Autor:
Eric S, Luth, Irina G, Stavrovskaya
Publikováno v:
Methods in molecular biology (Clifton, N.J.). 1948
Accumulation of misfolded αSyn and mitochondrial dysfunction are central features of Parkinson's disease. Growing evidence points to a relationship between these two phenomena as oligomeric α-synuclein (αSyn) can interact with mitochondria and imp
Autor:
Eric S. Luth, Irina G. Stavrovskaya
Publikováno v:
Methods in Molecular Biology ISBN: 9781493991235
Accumulation of misfolded αSyn and mitochondrial dysfunction are central features of Parkinson's disease. Growing evidence points to a relationship between these two phenomena as oligomeric α-synuclein (αSyn) can interact with mitochondria and imp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a76da2f3a28aa75161c1da30c2dfea8b
https://doi.org/10.1007/978-1-4939-9124-2_14
https://doi.org/10.1007/978-1-4939-9124-2_14
Autor:
Julia Hofer, Peter Juo, Molly Hodul, Eric S. Luth, Kaitlin Markoja, Carmino Riccio, Bethany J. Rennich
Publikováno v:
PLoS Genetics
PLoS Genetics, Vol 17, Iss 2, p e1009375 (2021)
PLoS Genetics, Vol 17, Iss 2, p e1009375 (2021)
Several intracellular trafficking pathways contribute to the regulation of AMPA receptor (AMPAR) levels at synapses and the control of synaptic strength. While much has been learned about these intracellular trafficking pathways, a major challenge is
Autor:
Peter Juo, Katherine Watters, Kelsey Jones, Julia Hofer, Eric S. Luth, Lidia Park, Irene Nguyen
Publikováno v:
PLoS ONE
PLoS ONE, Vol 16, Iss 2, p e0245587 (2021)
PLoS ONE, Vol 16, Iss 2, p e0245587 (2021)
Regulation of AMPA-type glutamate receptor (AMPAR) expression and function alters synaptic strength and is a major mechanism underlying synaptic plasticity. Although transcription is required for some forms of synaptic plasticity, the transcription f