Zobrazeno 1 - 10
of 64
pro vyhledávání: '"Enrique J. Sánchez Pozzi"'
Autor:
Viviana A. Catania, PhD, Enrique J. Sánchez Pozzi, PhD, Marcelo G. Luquita, PhD, María L. Ruiz, PhD, Silvina S.M. Villanueva, PhD, Brett Jones, PhD, Aldo D. Mottino, PhD
Publikováno v:
Annals of Hepatology, Vol 3, Iss 1, Pp 11-17 (2004)
Treatment of experimental animals with prototypical enzyme inducers represents a useful tool to characterize the role of different isozymes in drug metabolism and to improve our knowledge on factors regulating their synthesis at the transcriptional l
Externí odkaz:
https://doaj.org/article/9298b06b9de04b349e5cd91fef099c5a
Publikováno v:
Antioxidants & Redox Signaling. 35:808-831
Significance: Most hepatopathies are primarily or secondarily cholestatic in nature. Oxidative stress (OS) is a frequent trait among them, and impairs the machinery to generate bile by triggering e...
Autor:
Gisel Sabrina Miszczuk, Virginia Soledad Schuck, Fernando A. Crocenzi, Enrique J. Sánchez Pozzi, Nadia Ciriaci, Anabela Carolina Medeot, Romina Belén Andermatten, María Laura Ruiz, Ismael Ricardo Barosso, Marcelo G. Roma, María Valeria Razori
Publikováno v:
CONICET Digital (CONICET)
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
TNFα is a cytokine whose levels are increased in inflammatory pathologies that are associated with cholestasis. Endocytic internalization of Abcc2 (multidrug resistance-associated protein 2), a canalicular transporter of organic anions that is impli
Autor:
Romina Belén Andermatten, María Laura Ruiz, Cecilia Lorena Basiglio, Paula María Maidagan, Nadia Ciriaci, Enrique J. Sánchez Pozzi, Ismael Ricardo Barosso, María Valeria Razori, Marcelo G. Roma, Pamela Lucía Martín
Publikováno v:
CONICET Digital (CONICET)
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
Aims: Lipopolysaccharide (LPS) induces inflammatory cholestasis by impairing expression, localization, and function of carriers involved in bile formation, e.g. bile salt export pump (Bsep) and multidrug resistance-associated protein 2 (Mrp2). A spec
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8865b4c1d6be3c72ed1c39a0f84a57a1
https://linkinghub.elsevier.com/retrieve/pii/S002432052031105X
https://linkinghub.elsevier.com/retrieve/pii/S002432052031105X
Autor:
Ismael R Barosso, Andrés E Zucchetti, Andrea C Boaglio, M Cecilia Larocca, Diego R Taborda, Marcelo G Luquita, Marcelo G Roma, Fernando A Crocenzi, Enrique J Sánchez Pozzi
Publikováno v:
PLoS ONE, Vol 7, Iss 11, p e50711 (2012)
UnlabelledEstradiol 17ß-D-glucuronide (E17G) induces acute cholestasis in rat with endocytic internalization of the canalicular transporters bile salt export pump (Abcb11) and multidrug resistance-associated protein 2 (Abcc2). Classical protein kina
Externí odkaz:
https://doaj.org/article/84c3f8f9c4aa4c06b43872c5efbda6ac
Autor:
Andrea C Boaglio, Andrés E Zucchetti, Flavia D Toledo, Ismael R Barosso, Enrique J Sánchez Pozzi, Fernando A Crocenzi, Marcelo G Roma
Publikováno v:
PLoS ONE, Vol 7, Iss 11, p e49255 (2012)
The endogenous, cholestatic metabolite estradiol 17ß-D-glucuronide (E(2)17G) induces endocytic internalization of the canalicular transporters relevant to bile formation, Bsep and Mrp2. We evaluated here whether MAPKs are involved in this effect.ERK
Externí odkaz:
https://doaj.org/article/ff8c5f56b4ca4eb1b37e5813297fe616
Autor:
Pamela Lucía Martín, Enrique J. Sánchez Pozzi, Marcelo G. Roma, Cecilia Lorena Basiglio, María Laura Ruiz, Nadia Ciriaci, Romina Belén Andermatten, Ismael Ricardo Barosso, Paula María Maidagan, María Valeria Razori
Publikováno v:
CONICET Digital (CONICET)
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
Lipopolysaccharide (LPS) from Gram (-) bacteria induces inflammatory cholestasis by impairing the expression/localization of transporters involved in bile formation (e.g., Bsep, Mrp2). Therapeutic options for this disease are lacking. Ursodeoxycholic
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4b6b4f6dd6558af58ea0db23acdbd3f3
https://www.sciencedirect.com/science/article/abs/pii/S000629521930228X
https://www.sciencedirect.com/science/article/abs/pii/S000629521930228X
Autor:
Gisel Sabrina Miszczuk, Romina Belén Andermatten, Marcelo G. Roma, Nadia Ciriaci, Carlos Davio, Nicolás Di Siervi, María Valeria Razori, Enrique J. Sánchez Pozzi, Virginia Soledad Schuck, Ismael Ricardo Barosso, Anabela Carolina Medeot, Fernando A. Crocenzi
Publikováno v:
Archives of toxicology. 93(8)
Taurolithocholate (TLC) is a cholestatic bile salt that induces disinsertion of the canalicular transporter Abcc2 (Mrp2, multidrug resistance-associated protein 2). This internalization is mediated by different intracellular signaling proteins such a
Autor:
Marcelo G. Roma, Daniel E. Francés, Alejandra I. Martínez, Cecilia Lorena Basiglio, Enrique J. Sánchez Pozzi, Gerardo B. Pisani, Sandra Mónica María Arriaga, Paula Ceccatto, Pamela Lucía Martín, María Valeria Razori
Publikováno v:
Clinical science (London, England : 1979). 133(1)
We previously demonstrated in in vitro and ex vivo models that physiological concentrations of unconjugated bilirubin (BR) prevent oxidative stress (OS)-induced hepatocanalicular dysfunction and cholestasis. Here, we aimed to ascertain, in the whole
Autor:
Maria C. Larocca, Fernando A. Crocenzi, Enrique J. Sánchez Pozzi, Raúl A. Marinelli, Marcelo G. Roma, Julieta Marrone, Gisel Sabrina Miszczuk, Andrea Carolina Boaglio, Ismael Ricardo Barosso
Publikováno v:
CONICET Digital (CONICET)
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
Impaired canalicular secretion due to increased endocytosis and intracellular retention of canalicular transporters such as BSEP and MRP2 is a main, common pathomechanism of cholestasis. Nevertheless, the mechanisms governing this process are unknown
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8f7fdd91d4ca46b81f1be95909dd87b5
https://www.sciencedirect.com/science/article/pii/S0925443918300164
https://www.sciencedirect.com/science/article/pii/S0925443918300164