Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Enrico Faldini"'
Publikováno v:
Acta Neuropathologica Communications, Vol 7, Iss 1, Pp 1-13 (2019)
Abstract Many mouse models of Alzheimer’s disease (AD) exhibit impairments in hippocampal long-term-potentiation (LTP), seemingly corroborating the strong correlation between synaptic loss and cognitive decline reported in human studies. In other A
Externí odkaz:
https://doaj.org/article/8368c7c16d2b4c169e58d858f4711a8b
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 10 (2016)
Depotentiation (DP) is a mechanism by which synapses that have recently undergone long-term potentiation (LTP) can reverse their synaptic strengthening within a short time-window after LTP induction. Group 1 metabotropic glutamate receptors (mGluRs)
Externí odkaz:
https://doaj.org/article/df1d5ae9d22f4ea292ab80343d94fb1e
Publikováno v:
Acta Neuropathologica Communications, Vol 7, Iss 1, Pp 1-13 (2019)
Acta Neuropathologica Communications
Acta Neuropathologica Communications, BioMed Central part of Springer Science, 2019, 7 (1), pp.202. ⟨10.1186/s40478-019-0813-4⟩
Acta Neuropathologica Communications, 2019, 7 (1), pp.202. ⟨10.1186/s40478-019-0813-4⟩
Acta Neuropathologica Communications
Acta Neuropathologica Communications, BioMed Central part of Springer Science, 2019, 7 (1), pp.202. ⟨10.1186/s40478-019-0813-4⟩
Acta Neuropathologica Communications, 2019, 7 (1), pp.202. ⟨10.1186/s40478-019-0813-4⟩
Many mouse models of Alzheimer's disease (AD) exhibit impairments in hippocampal long-term-potentiation (LTP), seemingly corroborating the strong correlation between synaptic loss and cognitive decline reported in human studies. In other AD mouse mod
Autor:
Volkmar Gieselmann, Rudi D'Hooge, Matthias Eckhardt, Renate Lüllmann-Rauch, Detlef Balschun, Stijn Stroobants, Enrico Faldini
Publikováno v:
Behavioural Brain Research. 222:309-314
Arylsulfatase A-deficient (ASA(-/-)) mice constitute an animal model for metachromatic leukodystrophy, a lysosomal storage disorder. We had previously examined the behavioural phenotype of these mice, but were unable to distinguish between proper cog
Publikováno v:
Frontiers in Systems Neuroscience, Vol 8 (2014)
Neurons have the ability to modify their structure and function which ultimately serves for learning (Abraham and Bear, 1996). Dendritic events provide a major contribution to such modifications. For example, natural and artificial patterns of affere
Autor:
Alicia Oshlack, John C. Mulley, Judith Blanz, Renate M Kalnins, Lata Vadlamudi, Jeremy D. Silver, Jim Stankovich, Gordon K. Smyth, Ke Wei Zhang, Marta A. Bayly, David A. Power, Danya F. Vears, Judy Savige, John P. Dowling, Richard A L Macdonell, Leanne M. Dibbens, Frederick Andermann, Rudi D'Hooge, Renate Lüllmann-Rauch, Samuel F. Berkovic, Eva Andermann, Marina Katerelos, Melanie Bahlo, Bree L. Hodgson, Enrico Faldini, Paul Saftig
Publikováno v:
The American Journal of Human Genetics. (3):673-684
Action myoclonus-renal failure syndrome (AMRF) is an autosomal-recessive disorder with the remarkable combination of focal glomerulosclerosis, frequently with glomerular collapse, and progressive myoclonus epilepsy associated with storage material in