Zobrazeno 1 - 10
of 73
pro vyhledávání: '"Engel, J N"'
Autor:
Elwell, C.1, Engel, J. N.1,2 Jengel@medicine.ucsf.edu
Publikováno v:
Cellular Microbiology. May2005, Vol. 7 Issue 5, p725-739. 15p.
Autor:
Elwell, C, Engel, J N
Publikováno v:
Elwell, C; & Engel, J N. (2005). Drosophila melanogaster S2 cells: a model system to study Chlamydia interaction with host cells. Cellular Microbiology, 7(5), 725-739. UC San Francisco: Retrieved from: http://www.escholarship.org/uc/item/486567qx
Chlamydia spp. are major causes of important human diseases, but dissecting the host-pathogen interactions has been hampered by the lack of bacterial genetics and the difficulty in carrying out forward genetic screens in mammalian hosts. RNA interfer
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od_______325::1ae8dc18f76c7734e84a2df038667f7d
http://www.escholarship.org/uc/item/486567qx
http://www.escholarship.org/uc/item/486567qx
Publikováno v:
Gao, L Y; Guo, S; McLaughlin, B; Morisaki, H; Engel, J N; & Brown, Eric J. (2004). A mycobacterial virulence gene cluster extending RD1 is required for cytolysis, bacterial spreading and ESAT-6 secretion. Molecular Microbiology, 53(6), 1677-1693. UC San Francisco: Retrieved from: http://www.escholarship.org/uc/item/60x7h7m7
Initiation and maintenance of infection by mycobacteria in susceptible hosts are not well understood. A screen of Mycobacterium marinum transposon mutant library led to isolation of eight mutants that failed to cause haemolysis, all of which had tran
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od_______325::d763d3f5f664d9e6c17bfce228558e1e
http://www.escholarship.org/uc/item/60x7h7m7
http://www.escholarship.org/uc/item/60x7h7m7
Publikováno v:
Kazmierczak, B I; Mostov, K; & Engel, J N. (2004). Epithelial cell polarity alters Rho-GTPase responses to Pseudomonas aeruginosa. Molecular Biology of the Cell, 15(2), 411-419. UC San Francisco: Retrieved from: http://www.escholarship.org/uc/item/21r1g23h
Pseudomonas aeruginosa is an opportunistic human pathogen that preferentially infects damaged epithelial tissues. Previous studies have failed to distinguish whether the increased susceptibility of injured epithelium results from the loss of cell pol
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od_______325::ab233bd13b509cae819af8b1bcf43796
http://www.escholarship.org/uc/item/21r1g23h
http://www.escholarship.org/uc/item/21r1g23h
Akademický článek
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Publikováno v:
Annual Review of Microbiology; 2001, Vol. 55 Issue 1, p407, 29p
Publikováno v:
Cellular Microbiology; Feb2000, Vol. 3 Issue 2, p85-98, 14p
Publikováno v:
Molecular Biology of the Cell; May 2005, Vol. 16 Issue: 5 p2577-2585, 9p
Publikováno v:
Infection and Immunity; December 2004, Vol. 72 Issue: 12 p7367-7373, 7p
Autor:
Garrity-Ryan, L, Shafikhani, S, Balachandran, P, Nguyen, L, Oza, J, Jakobsen, T, Sargent, J, Fang, X, Cordwell, S, Matthay, M A, Engel, J N
Publikováno v:
Infection and Immunity; January 2004, Vol. 72 Issue: 1 p546-58, 13p