Novel role for p110β PI 3-Kinase in male fertility through regulation of androgen receptor activity in sertoli cells

Autor: Guillermet-Guibert, Julie, Smith, Lee B., Halet, Guillaume, Whitehead, Maria A., Pearce, Wayne, Rebourcet, Diane, León, Kelly, Crépieux, Pascale, Nock, Gemma, Strömstedt, Maria, Enerback, Malin, Chelala, Claude, Graupera, Mariona, Carroll, John, Cosulich, Sabina, Saunders, Philippa T. K., Huhtaniemi, Ilpo, Vanhaesebroeck, Bart

Publikováno v: Guillermet-Guibert, J, Smith, L B, Halet, G, Whitehead, M A, Pearce, W, Rebourcet, D, León, K, Crépieux, P, Nock, G, Strömstedt, M, Enerback, M, Chelala, C, Graupera, M, Carroll, J, Cosulich, S, Saunders, P T K, Huhtaniemi, I & Vanhaesebroeck, B 2015, ' Novel Role for p110β PI 3-Kinase in Male Fertility through Regulation of Androgen Receptor Activity in Sertoli Cells ', PLoS Genetics, vol. 11, no. 7, e1005304 . https://doi.org/10.1371/journal.pgen.1005304
PLoS Genetics
PLoS Genetics, 2015, 11 (7), pp.e1005304. ⟨10.1371/journal.pgen.1005304⟩
Recercat. Dipósit de la Recerca de Catalunya
instname
PLoS Genetics, Public Library of Science, 2015, 11 (7), pp.e1005304. ⟨10.1371/journal.pgen.1005304⟩
Dipòsit Digital de la UB
Universidad de Barcelona
PLoS Genetics, Vol 11, Iss 7, p e1005304 (2015)

The organismal roles of the ubiquitously expressed class I PI3K isoform p110β remain largely unknown. Using a new kinase-dead knockin mouse model that mimics constitutive pharmacological inactivation of p110β, we document that full inactivation of

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::af9a5a320333a3bd490870358a18d9f0
http://hdl.handle.net/10044/1/31606

Tento výsledek nelze pro nepřihlášené uživatele zobrazit.
K zobrazení výsledku je třeba se přihlásit.