Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Endothelial Cells/ metabolism/pathology"'
Autor:
Durré, Tania, Morfoisse, Florent, Erpicum, Charlotte, Ebroin, Marie, Blacher, Silvia, García-Caballero, Melissa, Deroanne, Christophe, Louis, Thomas, Balsat, Cédric, Van de Velde, Maureen, Kaijalainen, Seppo, Kridelka, Frederic, Engelholm, Lars, Struman, Ingrid, Alitalo, Kari, Behrendt, Niels, Paupert, Jenny, Noel, Agnès
Publikováno v:
Nature Communications, Vol 9, Iss 1, Pp 1-16 (2018)
Nature Communications
Nature communications, 9 (1
Nature Communications
Nature communications, 9 (1
The development of new lymphatic vessels occurs in many cancerous and inflammatory diseases through the binding of VEGF-C to its receptors, VEGFR-2 and VEGFR-3. The regulation of VEGFR-2/VEGFR-3 heterodimerisation and its downstream signaling in lymp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::1ba0b09e82a02f5e1a6abc11b5825577
https://doaj.org/article/38a29c790f274d7296f82886c67f9d1c
https://doaj.org/article/38a29c790f274d7296f82886c67f9d1c
Autor:
F.E.J. Coenjaerts, Brenda R. Kwak, Marc Chanson, Marc Bacchetta, Lucile Miquerol, Isabelle Roth, Zhihong Yang, T. A. B. van Veen, Urban Deutsch, Christos Chadjichristos, Habo J. Jongsma, M.J.A. van Kempen, M. Z. Richani Sarieddine, Tecla Dudez, K.E.L. Scheckenbach, Hema Viswambharan, Bernard Foglia, C. de Wit
Publikováno v:
Circulation
Circulation, 2010, 121 (1), pp.123-131. ⟨10.1161/CIRCULATIONAHA.109.867176⟩
Circulation, American Heart Association, 2010, 121 (1), pp.123-131
Circulation, Vol. 121, No 1 (2010) pp. 123-131
Circulation, 2010, 121 (1), pp.123-131. ⟨10.1161/CIRCULATIONAHA.109.867176⟩
Circulation, American Heart Association, 2010, 121 (1), pp.123-131
Circulation, Vol. 121, No 1 (2010) pp. 123-131
Background— Endothelial dysfunction is the initiating event of atherosclerosis. The expression of connexin40 (Cx40), an endothelial gap junction protein, is decreased during atherogenesis. In the present report, we sought to determine whether Cx40
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7bc33fba1a1aaaf799eda2172df5013a
https://hal.science/hal-00566980
https://hal.science/hal-00566980
Activation of the Rho GTPase Cdc42 has been shown in endothelial cell monolayers to prevent disassembly of interendothelial junctions and the increase in endothelial permeability. Here, we addressed the in vivo role of Cdc42 activity in mediating end
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od_____10561::dd91e9522948f11990ece10a1177291f
http://olympias.lib.uoi.gr/jspui/handle/123456789/22710
http://olympias.lib.uoi.gr/jspui/handle/123456789/22710
Autor:
Beat A. Imhof, Alain Vonlaufen, Catherine M. Pastor, Michel Aurrand-Lions, Jean-Louis Frossard, Antoine Hadengue, Chrystelle Lamagna
Publikováno v:
The Journal of Pathology, Vol. 209, No 4 (2006) pp. 540-548
The recruitment of inflammatory cells contributes significantly to tissue injury in acute pancreatitis. This process implies several molecular interactions between circulating and endothelial cells. The adhesion molecule junctional adhesion molecule
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b405c73946e0c05388cbb593b98b9183
https://pubmed.ncbi.nlm.nih.gov/16767690
https://pubmed.ncbi.nlm.nih.gov/16767690