Zobrazeno 1 - 10
of 22
pro vyhledávání: '"Emmanuel Labrada"'
Autor:
Carrie Sheeler, Emmanuel Labrada, Lisa Duvick, Leslie M. Thompson, Ying Zhang, Harry T. Orr, Marija Cvetanovic
Publikováno v:
Neurobiology of Disease, Vol 201, Iss , Pp 106673- (2024)
Spinocerebellar ataxia type 1 (SCA1) is a dominantly inherited and lethal neurodegenerative disease caused by the abnormal expansion of CAG repeats in the ATAXIN-1 (ATXN1) gene. Pathological studies identified dysfunction and loss of motor neurons (M
Externí odkaz:
https://doaj.org/article/340115b17b0e415f9876648f335e0074
Autor:
Katherine Hamel, Emmanuel Labrada Moncada, Carrie Sheeler, Juao-Guilherme Rosa, Stephen Gilliat, Ying Zhang, Marija Cvetanovic
Publikováno v:
Neurobiology of Disease, Vol 197, Iss , Pp 106530- (2024)
Summary: Heterogeneity is one of the key features of the healthy brain and selective vulnerability characterizes many, if not all, neurodegenerative diseases. While cerebellum contains majority of brain cells, neither its heterogeneity nor selective
Externí odkaz:
https://doaj.org/article/ea7a2f5bf6fe4de58c2caab364bd2d0d
Autor:
Alyssa Soles, Adem Selimovic, Kaelin Sbrocco, Ferris Ghannoum, Katherine Hamel, Emmanuel Labrada Moncada, Stephen Gilliat, Marija Cvetanovic
Publikováno v:
International Journal of Molecular Sciences, Vol 24, Iss 8, p 7049 (2023)
The extracellular matrix (ECM) surrounds cells in the brain, providing structural and functional support. Emerging studies demonstrate that the ECM plays important roles during development, in the healthy adult brain, and in brain diseases. The aim o
Externí odkaz:
https://doaj.org/article/9161b2d8d77248489da5d0deb8dfa4d4
Autor:
Hamel, Katherine, Moncada, Emmanuel Labrada, Sheeler, Carrie, Rosa, Juao-Guilherme, Gilliat, Stephen, Zhang, Ying, Cvetanovic, Marija
Publikováno v:
In Neurobiology of Disease July 2024 197
Akademický článek
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Autor:
Daniel Reyes-Haro, Francisco Emmanuel Labrada-Moncada, Durairaj Ragu Varman, Janina Krüger, Teresa Morales, Ricardo Miledi, Ataúlfo Martínez-Torres
Publikováno v:
Neural Plasticity, Vol 2016 (2016)
Anorexia nervosa is an eating disorder observed primarily in young women. The neurobiology of the disorder is unknown but recently magnetic resonance imaging showed a volume reduction of the hippocampus in anorexic patients. Dehydration-induced anore
Externí odkaz:
https://doaj.org/article/8a5db89e23634cd4bd47ccc91799fa1d
Autor:
Daniel Reyes-Haro, Francisco Emmanuel Labrada-Moncada, Ricardo Miledi, Ataúlfo Martínez-Torres
Publikováno v:
Neural Plasticity, Vol 2015 (2015)
Anorexia nervosa is an eating disorder associated with severe weight loss as a consequence of voluntary food intake avoidance. Animal models such as dehydration-induced anorexia (DIA) mimic core features of the disorder, including voluntary reduction
Externí odkaz:
https://doaj.org/article/cb110eef4144478fa93edd93b0f0165a
Akademický článek
Tento výsledek nelze pro nepřihlášené uživatele zobrazit.
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Publikováno v:
Neuroscience. 433
The cerebellum is involved in the coordination of movement. Its cellular composition is dominated by GABAergic neuronal types, and glial cells are known to express functional receptors. GABAergic signaling regulates cell proliferation, differentiatio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::7e37834bb904645080e677115821b5e8
https://pubmed.ncbi.nlm.nih.gov/32171821
https://pubmed.ncbi.nlm.nih.gov/32171821
Autor:
Durairaj Ragu-Varman, Ataúlfo Martínez-Torres, Mayra Macedo-Mendoza, Pamela Reyes-Ortega, Teresa Morales, Francisco Emmanuel Labrada-Moncada, Daniel Reyes-Haro
Publikováno v:
Behavioural brain research. 363
Anorexia by osmotic dehydration is an adaptive response to hypernatremia and hyperosmolaemia induced by ingestion of a hypertonic solution. Dehydration-induced anorexia (DIA) reproduces weight loss and avoidance of food, despite its availability. By
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bfb99b1a2f406375329839c9d72a0207
https://pubmed.ncbi.nlm.nih.gov/30690107
https://pubmed.ncbi.nlm.nih.gov/30690107