Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Emily Zifa"'
Autor:
Emily Zifa, Sotirios I. Sinis, Ilias Dimeas, Foteini Bardaka, Aglaia Pozantzi, Zoe Daniil, Konstantinos I. Gourgoulianis, Konstantina Rossi, Andreas Mouikis, Foteini Malli, Anna-Maria Psarra
Publikováno v:
Idiopathic interstitial pneumonias.
Autor:
Foteini Malli, Fotini Bardaka, Zissis Mamuris, Eugenios Bouzetos, Anna-Maria Psara, Emily Zifa, Maria Fouka, Ourania S. Kotsiou, Zoe Daniil, Helen Panagiotidou, Konstantinos I. Gourgoulianis, Katerina M. Antoniou
Publikováno v:
Sarcoidosis and other granulomatous ILD/DPLD.
Background: Complex I (NADH-ubiquinone oxidoreductase) is the largest enzyme of the respiratory electron transport chain in mitochondria, central to ATP production, mtDNA protein translation, membrane protein insertion, and stabilization. It is also
Autor:
Foteini Malli, Konstantinos I. Gourgoulianis, Zissis Mamuris, Alexandros Grammatikopoulos, Ourania S. Kotsiou, Emily Zifa, Zoe Daniil, Eirini Vasarmidi, Sotiria Peletidou, Katerina M. Antoniou, Helen Gkika
Publikováno v:
Mitochondrion. 43
Mitochondrial reactive oxygen species production may lead to tissue injury associated with two respiratory disorders of unknown origin which are shared by common tissue fibrosis, IPF and sarcoidosis. Sequence analysis of 22 mt-tRNA genes and parts of
Autor:
Emily Zifa, Helen Leze, Helen Maridaki, Zissis Mamuris, Archontia Kaminari, Paschalis Theotokis, Efrosini Petsiava, Constantinos Stathopoulos
Publikováno v:
Mitochondrion. 8:229-236
We describe a novel mutation in human mitochondrial NADH dehydrogenase 1 gene (ND1), a G to A transition at nucleotide position 3337, which is co-segregated with two known mutations in tRNALeu(CUN) A12308G and tRNAThr C15946T. These mutations were de
Autor:
Eleutheria Skoumi, Marini Terzenidou, Konstantinos Kostikas, Kostantinos Papadimitriou, Maria Stavrou, Konstantinos I. Gourgoulianis, Emily Zifa, Zissis Mamuris, Vasileios Bagiatis, Zoe Daniil
Publikováno v:
Molecular biology reports. 39(4)
A number of studies suggest that mitochondrial dysfunction plays a role in the pathogenesis of asthma. To shed light for the first time on the role of the mitochondrial genome in the etiology of asthma we analyzed the mitochondrial tRNA genes and par