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pro vyhledávání: '"Emily M. Armstrong"'
Autor:
Guillaume Lobet, Emily M. Armstrong, Zaigham Shahzad, Anna Amtmann, Simon Rogers, Adrian Hills, Fabian Kellermeier
Publikováno v:
Plant physiology 177, 1368–1381 (2018). doi:10.1104/pp.18.00217
Plant Physiology, Vol. 1, no.1, pp.00217.2018 (2018)
Plant Physiology, Vol. 1, no.1, pp.00217.2018 (2018)
If we want to understand how the environment has shaped the appearance and behavior of living creatures we need to compare groups of individuals that differ in genetic make-up and environment experience. For complex phenotypic features, such as body
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cef5ecaae3f790b02ec2c26bc791d5dd
https://hdl.handle.net/2128/19927
https://hdl.handle.net/2128/19927
Publikováno v:
Journal of Pharmacy Practice. 27:243-252
Pregnancy is associated with an increased risk of venous thromboembolism (VTE), with a reported incidence ranging from 0.49 to 2 events per 1000 deliveries. Risk factors include advanced maternal age, obesity, smoking, and cesarian section. Women wit
Publikováno v:
Journal of Pharmacy Practice. 27:253-259
Although controversial, screening for thrombophilia has become common. Testing for antiphospholipid antibodies is indicated in order to guide treatment decisions if there is clinical suspicion for antiphospholipid syndrome. The utility of identifying
Publikováno v:
Hospital practice (1995). 42(4)
Inhaled bronchodilators remain a cornerstone of treatment for chronic obstructive pulmonary disease (COPD); current guidelines recommend initiating inhaled bronchodilators as either monotherapy or combination therapy depending on disease severity and
Publikováno v:
Journal of pharmacy practice. 27(3)
Acquired thrombophilia is associated with an increased risk of venous thromboembolism (VTE). Antiphospholipid syndrome (APS) is the most prevalent acquired thrombophilia and is associated with both venous and arterial thromboses. Human immunodeficien
Publikováno v:
Journal of pharmacy practice. 27(3)
Thrombophilia alters normal hemostasis, shifting the balance in favor of thrombus formation. Inherited conditions include factor V Leiden (FVL), prothrombin G20210A mutation, deficiencies in natural anticoagulants (antithrombin [AT], protein C, and p