Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Emily A, Roltsch Hellard"'
Autor:
Cathy C. Y. Huang, Tengfei Ma, Emily A. Roltsch Hellard, Xuehua Wang, Amutha Selvamani, Jiayi Lu, Farida Sohrabji, Jun Wang
Publikováno v:
Scientific Reports, Vol 7, Iss 1, Pp 1-13 (2017)
Abstract Excessive alcohol consumption is a known risk factor for stroke, but the effect of stroke on alcohol intake is unknown. The dorsomedial striatum (DMS) and midbrain areas of the nigrostriatal circuit are critically associated to stroke and al
Externí odkaz:
https://doaj.org/article/be8a2e2935c647aeacf438e8fd4610b1
Autor:
Xiaowen Zhuang, Jun Wang, E. K. Williams, Bradley Jones, Tengfei Ma, Jiayi Lu, Swetha Jayavelu, Annalise Binette, Emily A. Roltsch Hellard
Publikováno v:
Neuropharmacology
Alcohol consumption alters glutamatergic transmission in many brain regions, including the dorsomedial striatum (DMS); this aberrant plasticity is thought to be responsible for alcohol-seeking behavior. Recent studies reported that alcohol induced su
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d305f378ad4ac68f1de15c20891318f5
https://pubmed.ncbi.nlm.nih.gov/31121177
https://pubmed.ncbi.nlm.nih.gov/31121177
Autor:
Annie M. Whitaker, Christy A. Itoga, Brandon A. Baiamonte, Heather N. Richardson, Brittni B. Baynes, Emily A. Roltsch Hellard, Yi-Ling Lu, Allyson L. Schreiber, Nicholas W. Gilpin
Publikováno v:
Neuropsychopharmacology. 41:2463-2472
Hyperalgesia is an exaggerated response to noxious stimuli produced by peripheral or central plasticity. Stress modifies nociception, and humans with post-traumatic stress disorder (PTSD) exhibit co-morbid chronic pain and amygdala dysregulation. Pre
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::24686780846a74b6e55d01f0988a4469
https://doi.org/10.1038/npp.2016.44
https://doi.org/10.1038/npp.2016.44
Publikováno v:
Addiction Biology. 22:692-701
Humans diagnosed with alcohol use disorder are more sensitive to painful stimuli during withdrawal, which suggests that excessive alcohol drinking worsens pain outcomes. Alcohol-dependent rats exhibit increases in nociceptive sensitivity during withd
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::6d796e8873ffdb2eb748cb796953c6a3
https://doi.org/10.1111/adb.12360
https://doi.org/10.1111/adb.12360
Autor:
Cathy C.Y. Huang, Jiayi Lu, Emily A. Roltsch Hellard, Xinsheng Gao, Jun-Yuan Ji, Yifeng Cheng, Xuehua Wang, Xiao Yan Wei, Jun Wang, Tengfei Ma
Publikováno v:
Nature neuroscience
Addiction is proposed to arise from alterations in synaptic strength via mechanisms of long-term potentiation (LTP) and depression (LTD). However, the causality between these synaptic processes and addictive behaviors is difficult to demonstrate. Her
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ad16351dddfc90e5e5046d3097eef2b6
https://pubmed.ncbi.nlm.nih.gov/29434375
https://pubmed.ncbi.nlm.nih.gov/29434375
Autor:
Dorit Ron, Yifeng Cheng, Hannah Gil, Jun Wang, Xuehua Wang, Tengfei Ma, Emily A. Roltsch Hellard, Sami Ben Hamida
Publikováno v:
The Journal of neuroscience : the official journal of the Society for Neuroscience. 35(33)
Addiction is thought to be a maladaptive form of learning and memory caused by drug-evoked aberrant synaptic plasticity. We previously showed that alcohol facilitates synaptic plasticity in the dorsomedial striatum (DMS), a brain region that drives g
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cb04e551bec15311959ec6139e7cb6f7
https://pubmed.ncbi.nlm.nih.gov/26290240
https://pubmed.ncbi.nlm.nih.gov/26290240
Publikováno v:
Addiction biology. 22(3)
Humans diagnosed with alcohol use disorder are more sensitive to painful stimuli during withdrawal, which suggests that excessive alcohol drinking worsens pain outcomes. Alcohol-dependent rats exhibit increases in nociceptive sensitivity during withd
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::48f54f7d1e174037d18ef991c0a7b251
https://pubmed.ncbi.nlm.nih.gov/26804193
https://pubmed.ncbi.nlm.nih.gov/26804193