Zobrazeno 1 - 10
of 23
pro vyhledávání: '"Elzbieta Sucajtys"'
Autor:
Elzbieta Sucajtys-Szulc, Alicja Debska-Slizien, Boleslaw Rutkowski, Ryszard Milczarek, Marek Szolkiewicz, Julian Swierczynski, Ryszard Tomasz Smolenski
Publikováno v:
International Journal of Molecular Sciences; Volume 24; Issue 6; Pages: 5733
Chronic kidney disease (CKD) is associated with elevated plasma fibrinogen concentration. However, the underlying molecular mechanism for elevated plasma fibrinogen concentration in CKD patients has not yet been clarified. We recently found that HNF1
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b891daf634163113eb77d9045d629252
https://doi.org/10.3390/ijms24065733
https://doi.org/10.3390/ijms24065733
Autor:
Ryszard Milczarek, Paulina Mierzejewska, Marcin Serocki, Barbara Kutryb-Zajac, Rafal Bartoszewski, Agnieszka Jasztal, Patrycja Koszałka, Alicja Bulinska, Stefan Chlopicki, Patrycja Jablonska, Ewa M. Slominska, Ryszard T. Smolenski, Elzbieta Sucajtys-Szulc, Magdalena A. Zabielska
Publikováno v:
Journal of Molecular and Cellular Cardiology. 128:62-76
Vascular inflammation is an important factor in the pathophysiology of cardiovascular diseases, such as atherosclerosis. Changes in the extracellular nucleotide and in particular adenosine catabolism may alter a chronic inflammation and endothelial a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f9d9b5aca7d523ec3326b122a4c06e4e
https://doi.org/10.1016/j.yjmcc.2019.01.004
https://doi.org/10.1016/j.yjmcc.2019.01.004
Autor:
Elzbieta Sucajtys-Szulc, Alicja Debska-Slizien, Boleslaw Rutkowski, Marek Szolkiewicz, Julian Swierczynski, Ryszard Tomasz Smolenski
Publikováno v:
International Journal of Molecular Sciences; Volume 23; Issue 16; Pages: 8883
Chronic kidney disease (CKD) is associated with low-grade inflammation that activates nuclear factor–κB (NF–κB), which upregulates the expression of numerous NF–κB responsive genes, including the genes encoding IL-6, ICAM-1, VCAM-1, and MCP-
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::81e233249426cd5648820112aa71a291
https://doi.org/10.3390/ijms23168883
https://doi.org/10.3390/ijms23168883
Publikováno v:
Molecular and Cellular Biochemistry
Studies designed to examine effects of fat mass reduction (including lipodystrophy and lipectomy) on human serum total and LDL-cholesterol concentrations are inconsistent. The purpose of this study was to examine effect of partial lipectomy in rats (
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b03197e8d27f522a64220c92af1a9cf7
https://doi.org/10.1007/s11010-018-3484-8
https://doi.org/10.1007/s11010-018-3484-8
Autor:
Iwona Pelikant-Malecka, Julian Swierczynski, Bolesław Rutkowski, Marek Szolkiewicz, Ryszard Milczarek, Tomasz Sledzinski, Alicja Debska-Slizien, Elzbieta Sucajtys-Szulc
Publikováno v:
Molecular and Cellular Biochemistry
Inflammation related to chronic kidney disease (CKD) is an important clinical problem. We recently determined that hepatocyte nuclear factor 1α (HNF1α) was upregulated in the livers of chronic renal failure (CRF) rats—experimental model of CKD. C
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7513e97c1f41906c628a7e7e73a19e63
https://doi.org/10.1007/s11010-018-3268-1
https://doi.org/10.1007/s11010-018-3268-1
Publikováno v:
Molecular and Cellular Biochemistry
Dyslipidemia commonly present in patients with chronic kidney disease (CKD) has been recently linked to increased proprotein convertase subtilisin/kexin type 9 (PCSK9) serum concentration. We tested a hypothesis that increased liver PCSK9 biosynthesi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5e452c003c58529aae88d52fa7061513
https://doi.org/10.1007/s11010-015-2590-0
https://doi.org/10.1007/s11010-015-2590-0
Publikováno v:
Endokrynologia Polska. 65:46-56
Wstep: Podanie progesteronu samicom szczura prowadzi do wzrostu ilości spozywanego przez nie pokarmu, jednak mechanizmy odpowiedzialne za ten efekt nie są do konca znane. Celem tej pracy bylo sprawdzenie, czy obserwowane pod wplywem progesteronu zm
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9d8ff0deef14aa3307f8dbe688871e11
https://doi.org/10.5603/ep.2014.0007
https://doi.org/10.5603/ep.2014.0007
Autor:
Bolesław Rutkowski, Julian Swierczynski, Joanna Blaszak, Elzbieta Sucajtys-Szulc, Marek Szolkiewicz, Marcin Konarzewski, Sławomir Lizakowski
Publikováno v:
American Journal of Nephrology. 40:157-163
Background: A high level of circulating PCSK9 binds to the LDL receptor, reduces its cell's surface density and leads to hypercholesterolemia. The aim of this study was to examine the circulating PCSK9 level in patients with kidney disease. Methods:
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::989341ac121da06f7c33a16d8b183134
https://doi.org/10.1159/000365935
https://doi.org/10.1159/000365935
Autor:
Michał Chmielewski, E Kossowska, Julian Swierczynski, Elzbieta Sucajtys-Szulc, Bolesław Rutkowski, Wojciech Boguslawski
Publikováno v:
Journal of Renal Nutrition. 18:448-455
Objective Enhanced liver cholesterol synthesis is present in experimental chronic renal failure (CRF), even though cholesterol concentrations in blood and liver are increased, suggesting that CRF results in disturbed cholesterolegenesis feedback regu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::20acfe54fd7d0fd37fda9b22c7362242
https://doi.org/10.1053/j.jrn.2008.04.005
https://doi.org/10.1053/j.jrn.2008.04.005
Publikováno v:
Atherosclerosis. 248
The aim of this study was to verify if an increase in Hnf1α gene expression could be a possible link between circulating proprotein convertase subtilisin/kexin type 9 (PCSK9) and TAGs concentrations in chronic renal failure (CRF).Rats underwent 5/6
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::934e91e711952726cd43512d1a379c39
https://pubmed.ncbi.nlm.nih.gov/26978583
https://pubmed.ncbi.nlm.nih.gov/26978583