Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Elodie Vimont"'
Autor:
Cecilia Patitucci, Juan Diego Hernández-Camacho, Elodie Vimont, Sonny Yde, Thomas Cokelaer, Thibault Chaze, Quentin Giai Gianetto, Mariette Matondo, Anastasia Gazi, Ivan Nemazanyy, David A. Stroud, Daniella H. Hock, Erminia Donnarumma, Timothy Wai
Publikováno v:
Nature Communications, Vol 14, Iss 1, Pp 1-21 (2023)
Abstract Hepatic steatosis is the result of imbalanced nutrient delivery and metabolism in the liver and is the first hallmark of Metabolic dysfunction-associated steatotic liver disease (MASLD). MASLD is the most common chronic liver disease and inv
Externí odkaz:
https://doaj.org/article/b09b45d1602f49a8a762f6a77dc40a33
Mitochondrial Fission Process 1 controls inner membrane integrity and protects against heart failure
Autor:
Erminia Donnarumma, Michael Kohlhaas, Elodie Vimont, Etienne Kornobis, Thibault Chaze, Quentin Giai Gianetto, Mariette Matondo, Maryse Moya-Nilges, Christoph Maack, Timothy Wai
Publikováno v:
Nature Communications, Vol 13, Iss 1, Pp 1-24 (2022)
Mitochondria power the beating heart. Here, Donnarumma et al. show that loss of the inner mitochondrial membrane protein MTFP1 in cardiomyocytes reduces bioenergetic efficiency and cell death resistance leading to heart failure in mice.
Externí odkaz:
https://doaj.org/article/e21337b4dba243f385e7018fb31873cd
Autor:
Emma Cretin, Priscilla Lopes, Elodie Vimont, Takashi Tatsuta, Thomas Langer, Anastasia Gazi, Martin Sachse, Patrick Yu‐Wai‐Man, Pascal Reynier, Timothy Wai
Publikováno v:
EMBO Molecular Medicine, Vol 13, Iss 6, Pp 1-29 (2021)
Abstract Mutations in OPA1 cause autosomal dominant optic atrophy (DOA) as well as DOA+, a phenotype characterized by more severe neurological deficits. OPA1 deficiency causes mitochondrial fragmentation and also disrupts cristae, respiration, mitoch
Externí odkaz:
https://doaj.org/article/0f7ab26593fd4ea9a7b36b1989bf8ee6
Publikováno v:
Archives of Cardiovascular Diseases Supplements. 15:210
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::5c635f686d29acd4d8b3084fb8208da6
https://doi.org/10.1016/j.acvdsp.2023.03.070
https://doi.org/10.1016/j.acvdsp.2023.03.070
Autor:
Timothy Wai, Christoph Maack, Maryse Moya-Nilges, Mariette Matondo, Quentin Giai Gianetto, Thibault Chaze, Etienne Kornobis, Elodie Vimont, Michael Kohlhaas, Erminia Donnarumma
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::40b02fe191e83db44578daa84f73fcaf
https://doi.org/10.15252/rc.2022838121
https://doi.org/10.15252/rc.2022838121
Autor:
Timothy Wai, Erminia Donnarumma, Christoph Maack, Thibault Chaze, Quentin Giai Gianetto, Mariette Matondo, Elodie Vimont, Etienne Kornobis, Michael Kohlhaas, Maryse Moya-Nilges
Mitochondria are paramount to the metabolism and survival of cardiomyocytes. Here we show that Mitochondrial Fission Process 1 (MTFP1) is essential for cardiac structure and function. Constitutive knockout of cardiomyocyte MTFP1 in mice resulted in a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ca769e02d74f988668b014b048320715
https://doi.org/10.1101/2021.10.21.465262
https://doi.org/10.1101/2021.10.21.465262