Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Ellis L, Ryan"'
Publikováno v:
Open Biology, Vol 7, Iss 8 (2017)
Fibroblast growth factor receptor 3–transforming acidic coiled-coil containing protein 3 (FGFR3–TACC3; FT3) is a gene fusion resulting from rearrangement of chromosome 4 that has been identified in many cancers including those of the urinary blad
Externí odkaz:
https://doaj.org/article/018d7ca9fadd4670913b4388ec7ad38c
Publikováno v:
Journal of Cell Science
article-version (VoR) Version of Record
article-version (VoR) Version of Record
A multiprotein complex containing TACC3, clathrin and other proteins has been implicated in mitotic spindle stability. To disrupt this complex in an anti-cancer context, we need to understand its composition and how it interacts with microtubules. In
Publikováno v:
Biomolecules, Vol 6, Iss 1, p 2 (2016)
RNA viruses are a genetically diverse group of pathogens that are responsible for some of the most prevalent and lethal human diseases. Numerous viruses introduce DNA damage and genetic instability in host cells during their lifecycles and some speci
Externí odkaz:
https://doaj.org/article/a69b150efbaa4254837101e6efc8fc55
Autor:
Nafiseh, Chalabi Hagkarim, Ellis L, Ryan, Philip J, Byrd, Robert, Hollingworth, Neil J, Shimwell, Angelo, Agathanggelou, Manon, Vavasseur, Viktoria, Kolbe, Thomas, Speiseder, Thomas, Dobner, Grant S, Stewart, Roger J, Grand
Publikováno v:
Journal of Virology
Infection by most DNA viruses activates a cellular DNA damage response (DDR), which may be to the detriment or advantage of the virus. In the case of adenoviruses, they neutralize antiviral effects of DDR activation by targeting a number of proteins
Autor:
Martin R. Higgs, Grant S. Stewart, Anastasia Zlatanou, John J. Reynolds, Simon J. Boulton, Edward S. Miller, Wojciech Niedzwiedz, Tatjana Stankovic, Alicja Winczura, Nicholas J. Davies, Valerie Borel, Andrew N. Blackford, Ellis L. Ryan, Jadwiga Nieminuszczy
Publikováno v:
Molecular cell. 59(3)
Recognition and repair of damaged replication forks are essential to maintain genome stability and are coordinated by the combined action of the Fanconi anemia and homologous recombination pathways. These pathways are vital to protect stalled replica