Zobrazeno 1 - 5
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pro vyhledávání: '"Elliot J. Crooks"'
Autor:
Jitika Rajpoot, Elliot J. Crooks, Brandon A. Irizarry, Ashley Amundson, William E. Van Nostrand, Steven O. Smith
Publikováno v:
Biochemistry. 61:1181-1198
Two distinct diseases are associated with the deposition of fibrillar amyloid-β (Aβ) peptides in the human brain in an age-dependent fashion. Alzheimer's disease is primarily associated with parenchymal plaque deposition of Aβ42, while cerebral am
Autor:
Feng Xu, William E. Van Nostrand, Michael D. Hoos, Helene Benveniste, John K. Robinson, Steven O. Smith, Elliot J. Crooks, Regina Kim, Judianne Davis, Hedok Lee, Dominique L. Popescu, Joshua Hatfield
Publikováno v:
The American Journal of Pathology. 188:2877-2889
Accumulation of fibrillar amyloid β protein in blood vessels of the brain, a condition known as cerebral amyloid angiopathy (CAA), is a common pathology of elderly individuals, a prominent comorbidity of Alzheimer disease, and a driver of vascular c
Autor:
Toru Kawakami, Elliot J. Crooks, Carlos Simmerling, Tiffany W Victor, Steven O. Smith, Lisa M. Miller, Feng Xu, William E. Van Nostrand, Hironobu Hojo, Martine Ziliox, Kelley Chiu, Brandon A. Irizarry
Publikováno v:
J Biol Chem
Cerebral amyloid angiopathy (CAA) is a vascular disorder that primarily involves deposition of the 40-residue-long β-amyloid peptide (Aβ40) in and along small blood vessels of the brain. CAA is often associated with Alzheimer's disease (AD), which
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2e40047e19b1326c72477601d1d5bb90
https://europepmc.org/articles/PMC7335782/
https://europepmc.org/articles/PMC7335782/
Publikováno v:
Methods in molecular biology (Clifton, N.J.). 1777
The amyloid-β (Aβ) peptides that form the amyloid fibrils associated with Alzheimer's disease are generated by sequential proteolysis of the amyloid precursor protein by β- and γ-secretase. The two predominant Aβ peptides, Aβ40 and Aβ42, diffe
Publikováno v:
Methods in Molecular Biology ISBN: 9781493978090
The amyloid-β (Aβ) peptides that form the amyloid fibrils associated with Alzheimer's disease are generated by sequential proteolysis of the amyloid precursor protein by β- and γ-secretase. The two predominant Aβ peptides, Aβ40 and Aβ42, diffe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a893991510524cc36bb2d4c66a095615
https://doi.org/10.1007/978-1-4939-7811-3_20
https://doi.org/10.1007/978-1-4939-7811-3_20