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of 4
pro vyhledávání: '"Eliza Peterson"'
Autor:
Anja Dokic, Eliza Peterson, Mario L. Arrieta-Ortiz, Min Pan, Alessandro Di Maio, Nitin Baliga, Apoorva Bhatt
Publikováno v:
The Cell Surface, Vol 7, Iss , Pp 100051- (2021)
A non-tuberculous mycobacterium, Mycobacterium abscessus is an emerging opportunistic pathogen associated with difficult to treat pulmonary infections, particularly in patients suffering from cystic fibrosis. It is capable of forming biofilms in vitr
Externí odkaz:
https://doaj.org/article/6129f7fb271d48d18014881c2165ba8e
Autor:
Perla Vega-Dominguez, Eliza Peterson, Min Pan, Alessandro Di Maio, Saumya Singh, Siva Umapathy, Deepak K. Saini, Nitin Baliga, Apoorva Bhatt
Publikováno v:
The Cell Surface, Vol 6, Iss , Pp 100043- (2020)
Mycobacterium chelonae is an environmental, non-tuberculous mycobacterial species, capable of causing infections in humans. Biofilm formation is a key strategy used by M. chelonae in colonising niches in the environment and in the host. We studied a
Externí odkaz:
https://doaj.org/article/219c122277894c0baf4d2d2f570cd520
Autor:
Sreetama Choudhury, Eliza Peterson, karolina Maciag, Ajay Suresh Akhade, Clifford Rostomily, Kevin Urdahl, Nitin Baliga, Naeha Subramanian
Publikováno v:
The Journal of Immunology. 206:97.19-97.19
Tuberculosis (TB) is a leading infectious disease that contributes to approximately 1.4 million deaths annually worldwide. In the present situation of the COVID-19 pandemic and disrupted healthcare services, mathematical modelling predicts that there
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ea6f032e04640a2beea6c9a23ef76c44
https://doi.org/10.4049/jimmunol.206.supp.97.19
https://doi.org/10.4049/jimmunol.206.supp.97.19
Autor:
Jared L Delahaye, Sara B Cohen, Eliza Peterson, Kristin Adams, Courtney R Plumlee, Christopher Plaisier, Nitin Baliga, Kevin B Urdahl
Publikováno v:
The Journal of Immunology. 204:156.3-156.3
During Mycobacterium tuberculosis (Mtb) infection, bacteria are inhaled into the lung where they first infect resident, alveolar macrophages (AM). The majority of the infection remains in AM until D14, when other cells, including recruited monocyte-d
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9d04696b66967c5304468e21c7d70418
https://doi.org/10.4049/jimmunol.204.supp.156.3
https://doi.org/10.4049/jimmunol.204.supp.156.3