Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Elisabeth L. Hahn"'
Publikováno v:
The Journal of Trauma: Injury, Infection, and Critical Care. 53:284-290
Background: The production of granulocyte colony-stimulating factor (G-CSF), the lineage specific essential regulator of neutrophil progenitor cell proliferation and differentiation, has been thought to be impaired in the setting of burn infection. T
Publikováno v:
Europe PubMed Central
This study evaluated macrophage expression of the stereospecific lactate transporter, MCT1, and the effects of lipopolysaccharide (LPS), tumor necrosis factor (TNFa), or nitric oxide (NO) on MCT1 mRNA and protein levels and lactate transporter activi
Publikováno v:
The Journal of Trauma: Injury, Infection, and Critical Care. 44:777-782
Prostaglandin E 2 (PGE 2 ) is significantly elevated in the plasma of septic or injured patients and is thought to be a component of the resultant immune suppression associated with augmented rates of infection and mortality. Many studies have examin
Publikováno v:
The Journal of trauma. 49(6)
Autor:
Hsin-Hsiung Tai, Elisabeth L. Hahn, Elizabeth J. Kovacs, Lisa A. Duffner, Douglas E. Faunce, Meredith S. Gregory
Publikováno v:
Cellular immunology. 205(2)
We have previously reported a macrophage-mediated gender difference in postburn immunosuppression, which was dependent upon elevated levels of circulating 17beta-estradiol (E(2)) and, in part, interleukin-6. Herein we examined the role of prostagland
Publikováno v:
The Journal of trauma. 47(6)
Objective: The aim of this study was to examine the relationship between prostaglandin synthesis and prostaglandin degradation in a model of burn injury with infection. Methods: Male B 2 D 6 F 1 mice were assigned to control, burn (16% dorsal scald b
Autor:
Keith D. Clancy, Cecilia Hofmann, Richard L. Gamelli, Elisabeth L. Hahn, Brett Christiansen, Kathryn Lorenz
Publikováno v:
The Journal of trauma. 42(2)
Burn injury and endotoxin lead to the development of a systemic inflammatory response. Because tumor necrosis factor-a (TNF-α) is a component of the proinflammatory response, we have determined the effect of burn injury and endotoxin in a murine mod
Publikováno v:
Shock. 9:4
Publikováno v:
Shock. 3:4
Publikováno v:
Shock. 3:49